Impaired AMPK Activity Drives Age-Associated Acute Lung Injury after Hemorrhage.
作者信息
Southern Brian D, Scheraga Rachel G, Olman Mitchell A
机构信息
1 Cleveland Clinic Respiratory Institute Lerner Research Institute Department of Pathobiology Cleveland, Ohio.
出版信息
Am J Respir Cell Mol Biol. 2017 May;56(5):553-555. doi: 10.1165/rcmb.2017-0023ED.
Abstract
摘要
相似文献
1
Impaired AMPK Activity Drives Age-Associated Acute Lung Injury after Hemorrhage.
Am J Respir Cell Mol Biol. 2017 May;56(5):553-555. doi: 10.1165/rcmb.2017-0023ED.
2
IL-10 administration attenuates pulmonary neutrophil infiltration and alters pulmonary iNOS activation following hemorrhagic shock.
Inflamm Res. 2009 Mar;58(3):170-4. doi: 10.1007/s00011-009-8116-z.
3
IL-10 deficiency augments acute lung but not liver injury in hemorrhagic shock.
Cytokine. 2009 Jan;45(1):26-31. doi: 10.1016/j.cyto.2008.10.004. Epub 2008 Nov 17.
4
Osthol attenuates neutrophilic oxidative stress and hemorrhagic shock-induced lung injury via inhibition of phosphodiesterase 4.
Free Radic Biol Med. 2015 Dec;89:387-400. doi: 10.1016/j.freeradbiomed.2015.08.008. Epub 2015 Nov 4.
5
Toll-like receptor 4 regulates heme oxygenase-1 expression after hemorrhagic shock induced acute lung injury in mice: requirement of p38 mitogen-activated protein kinase activation.
Shock. 2009 May;31(5):486-92. doi: 10.1097/SHK.0b013e318188f7e1.
6
Hydroxyethyl starch 6% (130/0.4) ameliorates acute lung injury in swine hemorrhagic shock.
Anesthesiology. 2010 Nov;113(5):1092-8. doi: 10.1097/ALN.0b013e3181f56029.
7
Deleterious effects of aggressive rapid crystalloid resuscitation on treatment of hyperinflammatory response and lung injury induced by hemorrhage in aging rats.
J Surg Res. 2014 Apr;187(2):587-95. doi: 10.1016/j.jss.2013.10.061. Epub 2013 Nov 7.
8
Calpain inhibitor I reduces the activation of nuclear factor-kappaB and organ injury/dysfunction in hemorrhagic shock.
FASEB J. 2001 Jan;15(1):171-186. doi: 10.1096/fj.99-0645com.
9
Critical role of toll-like receptor 4 in hypoxia-inducible factor 1α activation during trauma/hemorrhagic shocky induced acute lung injury after lymph infusion in mice.
Shock. 2014 Sep;42(3):271-8. doi: 10.1097/SHK.0000000000000212.
10
Influence of sex and age on morphological organ damage after hemorrhagic shock.
Shock. 2008 Jun;29(6):670-4. doi: 10.1097/shk.0b013e31815c3ea0.
引用本文的文献
1
Toll-Like Receptor 4 (TLR4) and AMPK Relevance in Cardiovascular Disease.
Adv Pharm Bull. 2023 Jan;13(1):36-47. doi: 10.34172/apb.2023.004. Epub 2021 Oct 10.
2
Plant polysaccharides with anti-lung injury effects as a potential therapeutic strategy for COVID-19.
Front Pharmacol. 2022 Oct 20;13:982893. doi: 10.3389/fphar.2022.982893. eCollection 2022.
3
Metformin Attenuates Silica-Induced Pulmonary Fibrosis by Activating Autophagy the AMPK-mTOR Signaling Pathway.
Front Pharmacol. 2021 Aug 9;12:719589. doi: 10.3389/fphar.2021.719589. eCollection 2021.
4
Metabolic Communication and Healthy Aging: Where Should We Focus Our Energy?
Dev Cell. 2020 Jul 20;54(2):196-211. doi: 10.1016/j.devcel.2020.06.011. Epub 2020 Jul 2.
5
PGC1α repression in IPF fibroblasts drives a pathologic metabolic, secretory and fibrogenic state.
Thorax. 2019 Aug;74(8):749-760. doi: 10.1136/thoraxjnl-2019-213064. Epub 2019 Jun 10.
本文引用的文献
1
Age-Dependent Changes in AMPK Metabolic Pathways in the Lung in a Mouse Model of Hemorrhagic Shock.
Am J Respir Cell Mol Biol. 2017 May;56(5):585-596. doi: 10.1165/rcmb.2016-0118OC.
2
Activation of AMPK attenuates LPS-induced acute lung injury by upregulation of PGC1α and SOD1.
Exp Ther Med. 2016 Sep;12(3):1551-1555. doi: 10.3892/etm.2016.3465. Epub 2016 Jun 17.
3
Regulation and function of AMPK in physiology and diseases.
Exp Mol Med. 2016 Jul 15;48(7):e245. doi: 10.1038/emm.2016.81.
4
Sirt1 and the Mitochondria.
Mol Cells. 2016 Feb;39(2):87-95. doi: 10.14348/molcells.2016.2318. Epub 2016 Feb 2.
5
Neutrophil-Derived MMP-8 Drives AMPK-Dependent Matrix Destruction in Human Pulmonary Tuberculosis.
PLoS Pathog. 2015 May 21;11(5):e1004917. doi: 10.1371/journal.ppat.1004917. eCollection 2015 May.
6
Advanced age is associated with worsened outcomes and a unique genomic response in severely injured patients with hemorrhagic shock.
Crit Care. 2015 Mar 4;19(1):77. doi: 10.1186/s13054-015-0788-x.
7
High CO2 levels cause skeletal muscle atrophy via AMP-activated kinase (AMPK), FoxO3a protein, and muscle-specific Ring finger protein 1 (MuRF1).
J Biol Chem. 2015 Apr 3;290(14):9183-94. doi: 10.1074/jbc.M114.625715. Epub 2015 Feb 17.
8
Role of AMP-Activated Protein Kinase (AMPK) in Smoking-Induced Lung Inflammation and Emphysema.
Tuberc Respir Dis (Seoul). 2015 Jan;78(1):8-17. doi: 10.4046/trd.2015.78.1.8. Epub 2015 Jan 29.
9
Adenosine monophosphate-activated protein kinase is required for pulmonary artery smooth muscle cell survival and the development of hypoxic pulmonary hypertension.
Am J Respir Cell Mol Biol. 2013 Oct;49(4):609-18. doi: 10.1165/rcmb.2012-0446OC.
10
Integrative analysis of complex cancer genomics and clinical profiles using the cBioPortal.
Sci Signal. 2013 Apr 2;6(269):pl1. doi: 10.1126/scisignal.2004088.
Zotero 插件