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胺碘酮所致甲状腺毒症——右心室流出道和左心室瘢痕性室性心动过速波动

Amiodarone Induced Thyrotoxicosis - Fluctuating RVOT and LV Scar VT.

作者信息

Pillarisetti Jayasree, Vanga Subba Reddy, Lakkireddy Dhanunjaya

机构信息

Mid America Cardiology @ University of Kansas Hospitals, Kansas City, KS.

出版信息

J Atr Fibrillation. 2013 Feb 12;5(5):342. doi: 10.4022/jafib.342. eCollection 2013 Feb-Mar.

Abstract

A 61 year old patient with non-ischemic cardiomyopathy and implantable cardioverter defibrillator presented with multiple shocks for ventricular tachycardia (VT). EKG revealed monomorphic sustained VT which was left bundle inferior axis that spontaneously changed into sustained VT which was right bundle superior axis. This was suggestive of an outflow tract VT transforming into a VT probably related to reentry from LV scar. The patient was transferred to our university for VT ablation. However, further investigation revealed amiodarone induced hyperthyroidism which was the cause of his ventricular tachycardia storm. Reversible causes of VT should be considered before proceeding with radiofrequency ablation.

摘要

一名61岁患有非缺血性心肌病且植入了心脏复律除颤器的患者因室性心动过速(VT)多次电击。心电图显示为单形性持续性室性心动过速,呈左束支下轴,后自发转变为右束支上轴的持续性室性心动过速。这提示流出道室性心动过速转变为可能与左心室瘢痕折返相关的室性心动过速。该患者被转至我校进行室性心动过速消融术。然而,进一步检查发现胺碘酮诱发的甲状腺功能亢进是其室性心动过速风暴的原因。在进行射频消融术前应考虑室性心动过速的可逆性病因。

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