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A Glitch in the Matrix: Aberrant Extracellular Matrix Proteolysis Contributes to Alcohol Seeking.

作者信息

Smith Alexander C W

机构信息

Department of Neuroscience, Icahn School of Medicine at Mount Sinai, New York, New York.

出版信息

Biol Psychiatry. 2017 Jun 1;81(11):900-902. doi: 10.1016/j.biopsych.2017.02.1181.

DOI:10.1016/j.biopsych.2017.02.1181
PMID:28502389
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC5742281/
Abstract
摘要

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Biol Psychiatry. 2017 Jun 1;81(11):907-917. doi: 10.1016/j.biopsych.2016.12.026. Epub 2017 Jan 5.
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Opposing mechanisms mediate morphine- and cocaine-induced generation of silent synapses.相反的机制介导吗啡和可卡因诱导的沉默突触的产生。
Nat Neurosci. 2016 Jul;19(7):915-25. doi: 10.1038/nn.4313. Epub 2016 May 30.
3
MMP-9 in translation: from molecule to brain physiology, pathology, and therapy.MMP-9的翻译：从分子到脑生理学、病理学及治疗
J Neurochem. 2016 Oct;139 Suppl 2:91-114. doi: 10.1111/jnc.13415. Epub 2016 Mar 21.
4
The tetrapartite synapse: Extracellular matrix remodeling contributes to corticoaccumbens plasticity underlying drug addiction.四联突触：细胞外基质重塑有助于药物成瘾背后的皮质-伏隔核可塑性。
Brain Res. 2015 Dec 2;1628(Pt A):29-39. doi: 10.1016/j.brainres.2015.03.027. Epub 2015 Mar 30.
5
Synaptically released matrix metalloproteinase activity in control of structural plasticity and the cell surface distribution of GluA1-AMPA receptors.突触释放的基质金属蛋白酶活性对结构可塑性和GluA1-AMPA受体细胞表面分布的调控
PLoS One. 2014 May 22;9(5):e98274. doi: 10.1371/journal.pone.0098274. eCollection 2014.
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Reward learning requires activity of matrix metalloproteinase-9 in the central amygdala.奖赏学习需要基质金属蛋白酶-9 在中枢杏仁核中的活性。
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