酒精与蛋白质缺乏对大鼠脑突触体（钠钾）-ATP酶的相互作用。
Interaction of alcohol and protein deficiency on rat brain synaptosomal (Na+-K+)-ATPase.
作者信息
Lalitha T, Ramakrishnan C V, Telang S D
机构信息
Department of Biochemistry, Faculty of Science, M.S. University of Baroda, India.
出版信息
Neurochem Res. 1988 Oct;13(10):963-6. doi: 10.1007/BF00970769.
PMID:2851113
Abstract
Administration of low amounts of ethanol for a prolonged period increases rat brain synaptosomal (Na+-K+)-ATPase activity, the increase being less in the protein deficient rats. The adaptive mechanism to offset the stress imposed by the continued presence of ethanol seems to be depressed by low plane of nutrition. In vivo and in vitro effects of ethanol on (Na+-K+)ATPase seems to be different.
摘要
长期给予少量乙醇会增加大鼠脑突触体（Na+-K+）-ATP酶的活性，而蛋白质缺乏的大鼠这种增加幅度较小。低营养水平似乎会抑制抵消乙醇持续存在所施加压力的适应性机制。乙醇对（Na+-K+）ATP酶的体内和体外作用似乎有所不同。
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