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Evidence that free radicals result in reperfusion injury in heart muscle.

作者信息

Weisfeldt M L, Zweier J, Ambrosio G, Becker L C, Flaherty J T

机构信息

Johns Hopkins Medical Institutions, Cardiology Division, Baltimore, Maryland 21205.

出版信息

Basic Life Sci. 1988;49:911-9. doi: 10.1007/978-1-4684-5568-7_149.

DOI:10.1007/978-1-4684-5568-7_149
PMID:2855014
Abstract
摘要

相似文献

1
Evidence that free radicals result in reperfusion injury in heart muscle.自由基导致心肌再灌注损伤的证据。
Basic Life Sci. 1988;49:911-9. doi: 10.1007/978-1-4684-5568-7_149.
2
Free radicals and myocardial reperfusion injury.自由基与心肌再灌注损伤
Br Med Bull. 1993 Jul;49(3):545-55. doi: 10.1093/oxfordjournals.bmb.a072629.
3
[Role of oxygen-radicals in myocardial reperfusion injury].
Nihon Naika Gakkai Zasshi. 1990 Sep 10;79(9):1219-24. doi: 10.2169/naika.79.1219.
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The role of neutrophils and free radicals in the ischemic-reperfused heart: why the confusion and controversy?中性粒细胞和自由基在缺血再灌注心脏中的作用：为何存在混淆和争议？
J Mol Cell Cardiol. 1989 Dec;21(12):1225-39. doi: 10.1016/0022-2828(89)90669-x.
5
[The role of oxygen-derived free radicals in ischemic heart damage].[氧衍生自由基在缺血性心脏损伤中的作用]
Kardiol Pol. 1988;31(12):786-98.
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Detection and measurement of free radical generation during cardiovascular injury.心血管损伤过程中自由基生成的检测与测量。
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7
Quantitative assessment of free-radical generation during ischemia and reperfusion in the isolated rabbit heart.
Coron Artery Dis. 1993 Dec;4(12):1093-6. doi: 10.1097/00019501-199312000-00009.
8
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Orv Hetil. 2015 Nov 22;156(47):1908-11. doi: 10.1556/650.2015.30304.
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Scavenging effect of Chinonin on NO and oxygen free radicals and its protective effect on the myocardium from the injury of ischemia-reperfusion.奎宁对一氧化氮和氧自由基的清除作用及其对心肌缺血再灌注损伤的保护作用。
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Soluble complement receptor type 1 inhibits the complement pathway and prevents contractile failure in the postischemic heart. Evidence that complement activation is required for neutrophil-mediated reperfusion injury.可溶性1型补体受体可抑制补体途径，并预防缺血后心脏的收缩功能衰竭。有证据表明补体激活是中性粒细胞介导的再灌注损伤所必需的。
Circulation. 1993 Dec;88(6):2812-26. doi: 10.1161/01.cir.88.6.2812.

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