Vinod Kateki, Panarelli Joseph F, Gentile Ronald C, Sidoti Paul A
Department of Ophthalmology, New York Eye and Ear Infirmary of Mount Sinai and Icahn School of Medicine at Mount Sinai, New York, NY.
J Glaucoma. 2017 Jul;26(7):669-672. doi: 10.1097/IJG.0000000000000694.
Vitreous occlusion of a glaucoma drainage implant (GDI) can lead to failure of the device and severely elevated intraocular pressure. The pathophysiology of tube obstruction is related to central and anterior displacement of vitreous that is drawn into and condenses within the proximal lumen of the tube. This can occur from days to years following GDI surgery. Successful management of vitreous-tube obstruction generally requires manual removal of the condensed vitreous plug with end-grasping forceps. This technique achieves reversal of tube blockage and restoration of GDI function. Amputation of the incarcerated vitreous alone with vitrectomy or neodymium:yttrium-aluminum-garnet vitreolysis does not consistently restore GDI function and risks persistent intraluminal tube obstruction.
青光眼引流植入物(GDI)的玻璃体阻塞可导致装置失效和眼压严重升高。导管阻塞的病理生理学与玻璃体的中央和前部移位有关,玻璃体被吸入导管近端管腔内并在其中凝结。这可发生在GDI手术后数天至数年。玻璃体-导管阻塞的成功处理通常需要用端部抓取钳手动移除凝结的玻璃体栓子。该技术可实现导管阻塞的逆转和GDI功能的恢复。仅通过玻璃体切除术或钕:钇铝石榴石玻璃体溶解术切除嵌顿的玻璃体并不能始终恢复GDI功能,且有持续管腔内阻塞的风险。