Activation of the human neutrophil superoxide-generating oxidase: studies in a reconstituted subcellular system.
作者信息
Clark R A, Volpp B D, Nauseef W M
机构信息
Department of Medicine, University of Iowa College of Medicine, Iowa City.
出版信息
Trans Am Clin Climatol Assoc. 1989;100:100-6.
Abstract
摘要
相似文献
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Activation of the human neutrophil superoxide-generating oxidase: studies in a reconstituted subcellular system.人类中性粒细胞超氧化物生成氧化酶的激活：在重组亚细胞系统中的研究
Trans Am Clin Climatol Assoc. 1989;100:100-6.
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Activation of human neutrophil nicotinamide adenine dinucleotide phosphate, reduced (triphosphopyridine nucleotide, reduced) oxidase by arachidonic acid in a cell-free system.在无细胞体系中，花生四烯酸对人中性粒细胞烟酰胺腺嘌呤二核苷酸磷酸（还原型）氧化酶（还原型三磷酸吡啶核苷酸氧化酶）的激活作用。
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3
The respiratory burst oxidase.呼吸爆发氧化酶。
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Classification of chronic granulomatous disease.慢性肉芽肿病的分类。
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[Activation mechanism of neutrophil NADPH oxidase complex--molecular pathology of chronic granulomatous disease].[中性粒细胞NADPH氧化酶复合物的激活机制——慢性肉芽肿病的分子病理学]
Tanpakushitsu Kakusan Koso. 1994 Nov;39(14):2284-96.
8
[NADPH oxidase and cell membrane].
Rinsho Byori. 1991 Sep;Suppl 91:140-9.
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Chronic granulomatous disease due to a defect in the cytosolic factor required for nicotinamide adenine dinucleotide phosphate oxidase activation.由于烟酰胺腺嘌呤二核苷酸磷酸氧化酶激活所需的胞质因子缺陷导致的慢性肉芽肿病。
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Heat shock in human neutrophils: superoxide generation is inhibited by a mechanism distinct from heat-denaturation of NADPH oxidase and is protected by heat shock proteins in thermotolerant cells.人类中性粒细胞中的热休克：超氧化物生成受一种不同于NADPH氧化酶热变性的机制抑制，且在耐热细胞中受到热休克蛋白的保护。
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本文引用的文献
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Subcellular localization of the b-cytochrome component of the human neutrophil microbicidal oxidase: translocation during activation.人类中性粒细胞杀菌氧化酶的β-细胞色素成分的亚细胞定位：激活过程中的易位
J Cell Biol. 1983 Jul;97(1):52-61. doi: 10.1083/jcb.97.1.52.
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Chronic granulomatous disease: a syndrome of phagocyte oxidase deficiencies.慢性肉芽肿病：一种吞噬细胞氧化酶缺乏综合征。
Medicine (Baltimore). 1983 Sep;62(5):286-309.
3
Stimulated neutrophils from patients with autosomal recessive chronic granulomatous disease fail to phosphorylate a Mr-44,000 protein.来自常染色体隐性慢性肉芽肿病患者的受刺激中性粒细胞无法使一种44,000道尔顿的蛋白质发生磷酸化。
Nature. 1985;316(6028):547-9. doi: 10.1038/316547a0.
4
Respiratory burst oxidase from human neutrophils: purification and some properties.人中性粒细胞呼吸爆发氧化酶：纯化及某些特性
Proc Natl Acad Sci U S A. 1985 May;82(10):3144-8. doi: 10.1073/pnas.82.10.3144.
5
The respiratory burst oxidase of human neutrophils. Further studies of the purified enzyme.人类中性粒细胞的呼吸爆发氧化酶。对纯化酶的进一步研究。
J Biol Chem. 1986 Oct 5;261(28):13247-51.
6
The respiratory burst oxidase of neutrophils. Separation of an FAD enzyme and its characterization.中性粒细胞的呼吸爆发氧化酶。一种黄素腺嘌呤二核苷酸（FAD）酶的分离及其特性鉴定。
J Biol Chem. 1987 Sep 5;262(25):12316-22.
7
The X-linked chronic granulomatous disease gene codes for the beta-chain of cytochrome b-245.X连锁慢性肉芽肿病基因编码细胞色素b - 245的β链。
Nature. 1987;327(6124):720-1. doi: 10.1038/327720a0.
8
The glycoprotein encoded by the X-linked chronic granulomatous disease locus is a component of the neutrophil cytochrome b complex.由X连锁慢性肉芽肿病基因座编码的糖蛋白是中性粒细胞细胞色素b复合物的一个组成部分。
Nature. 1987;327(6124):717-20. doi: 10.1038/327717a0.
9
Coregulation of NADPH oxidase activation and phosphorylation of a 48-kD protein(s) by a cytosolic factor defective in autosomal recessive chronic granulomatous disease.常染色体隐性慢性肉芽肿病中存在缺陷的胞质因子对NADPH氧化酶激活和一种48-kD蛋白磷酸化的共同调节。
J Clin Invest. 1988 May;81(5):1485-96. doi: 10.1172/JCI113480.
10
Relationship of protein phosphorylation to the activation of the respiratory burst in human neutrophils. Defects in the phosphorylation of a group of closely related 48-kDa proteins in two forms of chronic granulomatous disease.蛋白质磷酸化与人类中性粒细胞呼吸爆发激活之间的关系。两种形式的慢性肉芽肿病中一组密切相关的48 kDa蛋白质磷酸化的缺陷。
J Biol Chem. 1988 May 15;263(14):6777-82.
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