• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利：CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法，用中文像聊天一样搜索，搜遍4000万医学文献。AI智能推荐，让科研检索更轻松。

立即免费搜索

文件翻译

保留排版，准确专业，支持PDF/Word/PPT等文件格式，支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述，25分钟生成高质量综述，智能提取关键信息，辅助科研写作。

立即免费体验

Corrigendum to "KB004, a first in class monoclonal antibody targeting the receptor tyrosine kinase EphA3, in patients with advanced hematologic malignancies: Results from a phase 1 study" [Leuk. Res. 50 (Nov) (2016) 123-131. PubMed PMID: 27736729].

作者信息

Swords Ronan T, Greenberg Peter L, Wei Andrew H, Durrant Simon, Advani Anjali S, Hertzberg Mark S, Lewis Ian D, Rivera Gabriel, Gratzinger Dita, Fan Alice C, Felsher Dean W, Cortes Jorge E, Watts Justin M, Yarranton Geoff T, Walling Jackie M, Lancet Jeffrey E

机构信息

Leukemia Program, Sylvester Comprehensive Cancer Center, Miller School of Medicine, University of Miami, FL, United States.

Stanford Cancer Institute, Stanford, CA, United States.

出版信息

Leuk Res. 2017 Aug;59:65. doi: 10.1016/j.leukres.2017.02.007. Epub 2017 May 30.

DOI:10.1016/j.leukres.2017.02.007
PMID:28575698
Abstract
摘要

相似文献

1
Corrigendum to "KB004, a first in class monoclonal antibody targeting the receptor tyrosine kinase EphA3, in patients with advanced hematologic malignancies: Results from a phase 1 study" [Leuk. Res. 50 (Nov) (2016) 123-131. PubMed PMID: 27736729].
Leuk Res. 2017 Aug;59:65. doi: 10.1016/j.leukres.2017.02.007. Epub 2017 May 30.
2
KB004, a first in class monoclonal antibody targeting the receptor tyrosine kinase EphA3, in patients with advanced hematologic malignancies: Results from a phase 1 study.KB004，一种靶向受体酪氨酸激酶EphA3的首创单克隆抗体，用于晚期血液系统恶性肿瘤患者：1期研究结果。
Leuk Res. 2016 Nov;50:123-131. doi: 10.1016/j.leukres.2016.09.012. Epub 2016 Sep 28.
3
Comment on "KB004, a first in class monoclonal antibody targeting the receptor tyrosine kinase EphA3, in patients with advanced hematologic malignancies: Results from a phase 1 study".关于“KB004，一种靶向受体酪氨酸激酶EphA3的首创单克隆抗体，用于晚期血液系统恶性肿瘤患者：1期研究结果”的评论
Leuk Res. 2017 Apr;55:55-57. doi: 10.1016/j.leukres.2017.01.009. Epub 2017 Jan 9.
4
Copy number variations of EphA3 are associated with multiple types of hematologic malignancies.EphA3 的拷贝数变异与多种类型的血液系统恶性肿瘤有关。
Clin Lymphoma Myeloma Leuk. 2011 Feb;11(1):50-3. doi: 10.3816/CLML.2011.n.006.
5
EphA3 targeting reduces in vitro adhesion and invasion and in vivo growth and angiogenesis of multiple myeloma cells.EphA3 靶向减少多发性骨髓瘤细胞的体外黏附和侵袭以及体内生长和血管生成。
Cell Oncol (Dordr). 2017 Oct;40(5):483-496. doi: 10.1007/s13402-017-0338-4. Epub 2017 Jul 18.
6
Epha3 acts as proangiogenic factor in multiple myeloma.Epha3在多发性骨髓瘤中作为促血管生成因子发挥作用。
Oncotarget. 2017 May 23;8(21):34298-34309. doi: 10.18632/oncotarget.16100.
7
Nose-to-brain delivery of temozolomide-loaded PLGA nanoparticles functionalized with anti-EPHA3 for glioblastoma targeting.载替莫唑胺的 PLGA 纳米粒经抗 EphA3 修饰后经鼻腔递送至脑内用于脑胶质细胞瘤靶向治疗。
Drug Deliv. 2018 Nov;25(1):1634-1641. doi: 10.1080/10717544.2018.1494226.
8
Concurrent binding of anti-EphA3 antibody and ephrin-A5 amplifies EphA3 signaling and downstream responses: potential as EphA3-specific tumor-targeting reagents.抗EphA3抗体与ephrin-A5的同时结合增强EphA3信号传导及下游反应：作为EphA3特异性肿瘤靶向试剂的潜力。
Cancer Res. 2005 Aug 1;65(15):6745-54. doi: 10.1158/0008-5472.CAN-05-0758.
9
Corrigendum to "BCL11A expression in acute phase chronic myeloid leukemia" [Leuk. Res. 47 (2016) 88-92].《“慢性髓系白血病急性期的BCL11A表达”勘误》[《白血病研究》47卷（2016年）第88 - 92页]
Leuk Res. 2017 Jan;52:67. doi: 10.1016/j.leukres.2016.08.001. Epub 2016 Nov 21.
10
Loss of EphA3 Protein Expression Is Associated With Advanced TNM Stage in Clear-Cell Renal Cell Carcinoma.EphA3蛋白表达缺失与透明细胞肾细胞癌的晚期TNM分期相关。
Clin Genitourin Cancer. 2017 Apr;15(2):e169-e173. doi: 10.1016/j.clgc.2016.07.028. Epub 2016 Aug 8.

引用本文的文献

1
Loss of EphA7 Expression in Basal Cell Carcinoma by Hypermethylation of CpG Islands in the Promoter Region.抑癌基因 EphA7 在基底细胞癌中启动子区 CpG 岛高甲基化导致表达缺失。
Anal Cell Pathol (Amst). 2022 Jan 22;2022:4220786. doi: 10.1155/2022/4220786. eCollection 2022.