Urine Ammonium and Preclinical Acidosis in CKD.
作者信息
DuBose Thomas D
机构信息
Wake Forest School of Medicine; and Section of General Internal Medicine, Winston Salem, North Carolina
出版信息
J Am Soc Nephrol. 2017 Aug;28(8):2258-2260. doi: 10.1681/ASN.2017040470. Epub 2017 Jun 19.
Abstract
摘要
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Acidosis Activates Endoplasmic Reticulum Stress Pathways through GPR4 in Human Vascular Endothelial Cells.酸中毒通过人血管内皮细胞中的GPR4激活内质网应激途径。
Int J Mol Sci. 2017 Jan 27;18(2):278. doi: 10.3390/ijms18020278.
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Acid retention with reduced glomerular filtration rate increases urine biomarkers of kidney and bone injury.酸潴留伴肾小球滤过率降低会增加肾脏和骨骼损伤的尿液生物标志物。
Kidney Int. 2017 Apr;91(4):914-927. doi: 10.1016/j.kint.2016.10.023. Epub 2016 Dec 15.
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Int J Mol Sci. 2015 May 15;16(5):11055-86. doi: 10.3390/ijms160511055.
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High Dietary Acid Load Predicts ESRD among Adults with CKD.高膳食酸负荷可预测慢性肾脏病成人患者的终末期肾病。
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Treatment of metabolic acidosis in patients with stage 3 chronic kidney disease with fruits and vegetables or oral bicarbonate reduces urine angiotensinogen and preserves glomerular filtration rate.用水果和蔬菜或口服碳酸氢盐治疗 3 期慢性肾脏病患者的代谢性酸中毒可减少尿血管紧张素原并维持肾小球滤过率。
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Acid retention accompanies reduced GFR in humans and increases plasma levels of endothelin and aldosterone.在人类中，酸潴留伴随着肾小球滤过率（GFR）降低，并增加血浆内皮素和醛固酮水平。
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