Endocannabinoid metabolism in neurodegenerative diseases.
作者信息
Chen Chu
机构信息
Neuroscience Center of Excellence, School of Medicine, Louisiana State University Health Sciences Center, New Orleans, Louisiana 70112, USA.
出版信息
Neuroimmunol Neuroinflamm. 2016;3:268-270. doi: 10.20517/2347-8659.2016.46. Epub 2016 Dec 15.
Abstract
摘要
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本文引用的文献
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Traumatic Brain Injury as a Risk Factor for Alzheimer's Disease: Is Inflammatory Signaling a Key Player?创伤性脑损伤作为阿尔茨海默病的一个风险因素：炎症信号是关键因素吗？
Curr Alzheimer Res. 2016;13(7):730-8. doi: 10.2174/1567205013666160222110320.
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Traumatic brain injury: a risk factor for neurodegenerative diseases.创伤性脑损伤：神经退行性疾病的一个风险因素。
Rev Neurosci. 2016 Jan;27(1):93-100. doi: 10.1515/revneuro-2015-0017.
3
Inhibition of monoacylglycerol lipase prevents chronic traumatic encephalopathy-like neuropathology in a mouse model of repetitive mild closed head injury.抑制单酰甘油脂肪酶可预防重复性轻度闭合性颅脑损伤小鼠模型中的慢性创伤性脑病样神经病理学变化。
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4
Synaptic and cognitive improvements by inhibition of 2-AG metabolism are through upregulation of microRNA-188-3p in a mouse model of Alzheimer's disease.在阿尔茨海默病小鼠模型中，通过抑制2-花生四烯酸甘油（2-AG）代谢实现的突触和认知改善是通过上调微小RNA-188-3p来实现的。
J Neurosci. 2014 Nov 5;34(45):14919-33. doi: 10.1523/JNEUROSCI.1165-14.2014.
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Endocannabinoids in synaptic plasticity and neuroprotection.内源性大麻素在突触可塑性和神经保护中的作用
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Monoacylglycerol lipase is a therapeutic target for Alzheimer's disease.单酰甘油脂肪酶是阿尔茨海默病的治疗靶点。
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