Lung Interstitial Macrophages Redefined: It Is Not That Simple Anymore.
作者信息
Reddy Sekhar P, Mehta Dolly
机构信息
1 Departments of Pediatrics and Pharmacology University of Illinois at Chicago Chicago, Illinois.
出版信息
Am J Respir Cell Mol Biol. 2017 Aug;57(2):135-136. doi: 10.1165/rcmb.2017-0158ED.
Abstract
摘要
相似文献
1
Lung Interstitial Macrophages Redefined: It Is Not That Simple Anymore.肺间质巨噬细胞的重新定义：不再那么简单了。
Am J Respir Cell Mol Biol. 2017 Aug;57(2):135-136. doi: 10.1165/rcmb.2017-0158ED.
2
Monocyte-derived Alveolar Macrophages: The Dark Side of Lung Repair?单核细胞衍生的肺泡巨噬细胞：肺修复的阴暗面？
Am J Respir Cell Mol Biol. 2018 Jan;58(1):5-6. doi: 10.1165/rcmb.2017-0328ED.
3
Pretreatment with bone marrow-derived mesenchymal stromal cell-conditioned media confers pulmonary ischemic tolerance.骨髓源性间充质基质细胞条件培养基预处理可赋予肺缺血耐受性。
J Thorac Cardiovasc Surg. 2016 Mar;151(3):841-849. doi: 10.1016/j.jtcvs.2015.11.043. Epub 2015 Nov 30.
4
CD11b(+) Mononuclear Cells Mitigate Hyperoxia-Induced Lung Injury in Neonatal Mice.CD11b(+)单核细胞减轻新生小鼠高氧诱导的肺损伤。
Am J Respir Cell Mol Biol. 2016 Feb;54(2):273-83. doi: 10.1165/rcmb.2014-0395OC.
5
Effects of dexmedetomidine on the protection of hyperoxia-induced lung injury in newborn rats.右美托咪定对新生大鼠高氧诱导肺损伤的保护作用。
Int J Clin Exp Pathol. 2015 Jun 1;8(6):6466-73. eCollection 2015.
6
Double-Stranded RNA Interacts With Toll-Like Receptor 3 in Driving the Acute Inflammatory Response Following Lung Contusion.双链RNA在肺挫伤后驱动急性炎症反应过程中与Toll样受体3相互作用。
Crit Care Med. 2016 Nov;44(11):e1054-e1066. doi: 10.1097/CCM.0000000000001879.
7
The pathogenesis of bleomycin-induced lung injury in animals and its applicability to human idiopathic pulmonary fibrosis.博来霉素诱导的动物肺损伤的发病机制及其在人类特发性肺纤维化中的适用性。
Exp Lung Res. 2015 Mar;41(2):57-73. doi: 10.3109/01902148.2014.979516. Epub 2014 Dec 16.
8
Macrophages and inflammatory mediators in pulmonary injury induced by mustard vesicants.芥子气致肺损伤中的巨噬细胞与炎症介质
Ann N Y Acad Sci. 2016 Jun;1374(1):168-75. doi: 10.1111/nyas.13123. Epub 2016 Jun 28.
9
Differential toll-like receptor activation in lung ischemia reperfusion injury.肺缺血再灌注损伤中Toll样受体的差异性激活
J Thorac Cardiovasc Surg. 2015 Jun;149(6):1653-61. doi: 10.1016/j.jtcvs.2015.02.045. Epub 2015 Feb 28.
10
STAT4 knockout protects LPS-induced lung injury by increasing of MDSC and promoting of macrophage differentiation.STAT4基因敲除通过增加髓系来源的抑制细胞（MDSC）和促进巨噬细胞分化来保护脂多糖诱导的肺损伤。
Respir Physiol Neurobiol. 2016 Mar;223:16-22. doi: 10.1016/j.resp.2015.11.016. Epub 2015 Nov 28.
引用本文的文献
1
OC-STAMP is a potential biomarker and therapeutic target for Silicosis: an exploratory investigation.OC-STAMP是矽肺的潜在生物标志物和治疗靶点：一项探索性研究。
J Transl Med. 2025 Feb 21;23(1):214. doi: 10.1186/s12967-024-05981-3.
2
Resident macrophages of the lung and liver: The guardians of our tissues.肺和肝的固有巨噬细胞：组织的守护者。
Front Immunol. 2022 Nov 30;13:1029085. doi: 10.3389/fimmu.2022.1029085. eCollection 2022.
3
The emerging roles of interstitial macrophages in pulmonary fibrosis: A perspective from scRNA-seq analyses.间充质巨噬细胞在肺纤维化中的新兴作用：来自 scRNA-seq 分析的观点。
Front Immunol. 2022 Sep 22;13:923235. doi: 10.3389/fimmu.2022.923235. eCollection 2022.
4
Macrophage TLR4 and PAR2 Signaling: Role in Regulating Vascular Inflammatory Injury and Repair.巨噬细胞 TLR4 和 PAR2 信号通路：在调节血管炎症损伤和修复中的作用。
Front Immunol. 2020 Sep 18;11:2091. doi: 10.3389/fimmu.2020.02091. eCollection 2020.
5
Alveolar lipids in pulmonary disease. A review.肺部疾病中的肺泡脂质。综述。
Lipids Health Dis. 2020 Jun 3;19(1):122. doi: 10.1186/s12944-020-01278-8.
6
Quantitative biokinetics over a 28 day period of freshly generated, pristine, 20 nm titanium dioxide nanoparticle aerosols in healthy adult rats after a single two-hour inhalation exposure.健康成年大鼠单次两小时吸入暴露后 28 天内新生成的原始 20nm 二氧化钛纳米颗粒气溶胶的定量生物动力学。
Part Fibre Toxicol. 2019 Jul 9;16(1):29. doi: 10.1186/s12989-019-0303-7.
7
CD11b interstitial macrophages are required for ischemia-induced lung angiogenesis.缺血诱导的肺血管生成需要CD11b间质巨噬细胞。
Physiol Rep. 2018 Jun;6(11):e13721. doi: 10.14814/phy2.13721.
本文引用的文献
1
Three Unique Interstitial Macrophages in the Murine Lung at Steady State.稳态下小鼠肺中三种独特的间质巨噬细胞。
Am J Respir Cell Mol Biol. 2017 Jul;57(1):66-76. doi: 10.1165/rcmb.2016-0361OC.
2
Origin and functions of tissue macrophages.组织巨噬细胞的起源与功能。
Immunity. 2014 Jul 17;41(1):21-35. doi: 10.1016/j.immuni.2014.06.013.
3
In vivo characterization of alveolar and interstitial lung macrophages in rhesus macaques: implications for understanding lung disease in humans.体内鉴定恒河猴肺泡和肺间质巨噬细胞：对理解人类肺部疾病的启示。
J Immunol. 2014 Mar 15;192(6):2821-9. doi: 10.4049/jimmunol.1302269. Epub 2014 Feb 17.
4
Sessile alveolar macrophages communicate with alveolar epithelium to modulate immunity.无功能性孤立性肾上腺结节的 CT 诊断
Nature. 2014 Feb 27;506(7489):503-6. doi: 10.1038/nature12902. Epub 2014 Jan 19.
5
Alveolar macrophages: plasticity in a tissue-specific context.肺泡巨噬细胞：组织特异性环境中的可塑性。
Nat Rev Immunol. 2014 Feb;14(2):81-93. doi: 10.1038/nri3600. Epub 2014 Jan 21.
6
Gene-expression profiles and transcriptional regulatory pathways that underlie the identity and diversity of mouse tissue macrophages.小鼠组织巨噬细胞的特征和多样性所依赖的基因表达谱和转录调控途径。
Nat Immunol. 2012 Nov;13(11):1118-28. doi: 10.1038/ni.2419. Epub 2012 Sep 30.
7
Leukocyte compartments in the mouse lung: distinguishing between marginated, interstitial, and alveolar cells in response to injury.小鼠肺部的白细胞区室：损伤后区分边缘、间质和肺泡细胞。
J Immunol Methods. 2012 Jan 31;375(1-2):100-10. doi: 10.1016/j.jim.2011.09.013. Epub 2011 Oct 2.
8
Macrophage diversity enhances tumor progression and metastasis.巨噬细胞多样性增强肿瘤的进展和转移。
Cell. 2010 Apr 2;141(1):39-51. doi: 10.1016/j.cell.2010.03.014.
9
Lung interstitial macrophages alter dendritic cell functions to prevent airway allergy in mice.肺间质巨噬细胞改变树突状细胞功能，防止小鼠发生气道过敏。
J Clin Invest. 2009 Dec;119(12):3723-38. doi: 10.1172/JCI39717. Epub 2009 Nov 9.
10
Trophic macrophages in development and disease.发育和疾病中的营养性巨噬细胞。
Nat Rev Immunol. 2009 Apr;9(4):259-70. doi: 10.1038/nri2528.
Zotero 插件