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应激生存:正常细胞与恶性细胞中ATF4介导的应激反应调节

Surviving Stress: Modulation of ATF4-Mediated Stress Responses in Normal and Malignant Cells.

作者信息

Wortel Inge M N, van der Meer Laurens T, Kilberg Michael S, van Leeuwen Frank N

机构信息

Laboratory of Pediatric Oncology, Radboud Institute for Molecular Life Science, Radboud university medical center, Nijmegen, The Netherlands; Department of Tumor Immunology, Radboud Institute for Molecular Life Sciences, Radboud university medical center, Nijmegen, The Netherlands.

Laboratory of Pediatric Oncology, Radboud Institute for Molecular Life Science, Radboud university medical center, Nijmegen, The Netherlands.

出版信息

Trends Endocrinol Metab. 2017 Nov;28(11):794-806. doi: 10.1016/j.tem.2017.07.003. Epub 2017 Aug 7.

Abstract

Activating transcription factor 4 (ATF4) is a stress-induced transcription factor that is frequently upregulated in cancer cells. ATF4 controls the expression of a wide range of adaptive genes that allow cells to endure periods of stress, such as hypoxia or amino acid limitation. However, under persistent stress conditions, ATF4 promotes the induction of apoptosis. Recent advances point to a role for post-translational modifications (PTMs) and epigenetic mechanisms in balancing these pro- and anti-survival effects of ATF4. We review here how PTMs and epigenetic modifiers associated with ATF4 may be exploited by cancer cells to cope with cellular stress conditions that are intrinsically associated with tumor growth. Identification of mechanisms that modulate ATF4-mediated transcription and its effects on cellular metabolism may uncover new targets for cancer treatment.

摘要

激活转录因子4(ATF4)是一种应激诱导的转录因子,在癌细胞中经常上调。ATF4控制多种适应性基因的表达,使细胞能够耐受应激期,如缺氧或氨基酸限制。然而,在持续应激条件下,ATF4促进细胞凋亡的诱导。最近的进展表明,翻译后修饰(PTM)和表观遗传机制在平衡ATF4的这些促生存和抗生存效应中发挥作用。我们在此综述癌细胞如何利用与ATF4相关的PTM和表观遗传修饰因子来应对与肿瘤生长内在相关的细胞应激条件。鉴定调节ATF4介导的转录及其对细胞代谢影响的机制可能会揭示癌症治疗的新靶点。

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