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Dengue 2 virus inhibits in vitro megakaryocytic colony formation and induces apoptosis in thrombopoietin-inducible megakaryocytic differentiation from cord blood CD34+ cells.登革2型病毒抑制体外巨核细胞集落形成,并诱导脐带血CD34+细胞在血小板生成素诱导下的巨核细胞分化过程发生凋亡。
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Front Immunol. 2021 Apr 29;12:616394. doi: 10.3389/fimmu.2021.616394. eCollection 2021.
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Immunomodulatory Activities of L. Leaf Juice in a Non-Lethal, Symptomatic Dengue Mouse Model.莱氏叶汁在非致死性、有症状登革热小鼠模型中的免疫调节活性
Pathogens. 2021 Apr 21;10(5):501. doi: 10.3390/pathogens10050501.

本文引用的文献

1
Emerging Infectious Diseases.新发传染病
Adv Pediatr. 2017 Aug;64(1):27-71. doi: 10.1016/j.yapd.2017.04.002.
2
Suppressive effect of dengue virus envelope protein domain III on megakaryopoiesis.登革病毒包膜蛋白结构域 III 对巨核细胞生成的抑制作用。
Virulence. 2017 Nov 17;8(8):1719-1731. doi: 10.1080/21505594.2017.1343769. Epub 2017 Jul 20.
3
Anti-dengue virus nonstructural protein 1 antibodies contribute to platelet phagocytosis by macrophages.抗登革病毒非结构蛋白 1 抗体有助于巨噬细胞吞噬血小板。
Thromb Haemost. 2016 Mar;115(3):646-56. doi: 10.1160/TH15-06-0498. Epub 2015 Dec 3.
4
Thrombocytopenia in Dengue: Interrelationship between Virus and the Imbalance between Coagulation and Fibrinolysis and Inflammatory Mediators.登革热中的血小板减少症:病毒与凝血、纤维蛋白溶解及炎症介质失衡之间的相互关系
Mediators Inflamm. 2015;2015:313842. doi: 10.1155/2015/313842. Epub 2015 Apr 27.
5
Dengue virus binding and replication by platelets.登革病毒与血小板的结合及复制
Blood. 2015 Jul 16;126(3):378-85. doi: 10.1182/blood-2014-09-598029. Epub 2015 May 5.
6
WHO dengue case classification 2009 and its usefulness in practice: an expert consensus in the Americas.《2009年世界卫生组织登革热病例分类及其在实际中的效用:美洲地区专家共识》
Pathog Glob Health. 2015 Feb;109(1):19-25. doi: 10.1179/2047773215Y.0000000003. Epub 2015 Jan 28.
7
Loss of autophagy leads to failure in megakaryopoiesis, megakaryocyte differentiation, and thrombopoiesis in mice.自噬缺失会导致小鼠巨核细胞生成、巨核细胞分化及血小板生成出现障碍。
Exp Hematol. 2015 Jun;43(6):488-94. doi: 10.1016/j.exphem.2015.01.001. Epub 2015 Jan 13.
8
Virus interactions with endothelial cell receptors: implications for viral pathogenesis.病毒与内皮细胞受体的相互作用:对病毒发病机制的影响。
Curr Opin Virol. 2014 Aug;7:134-40. doi: 10.1016/j.coviro.2014.06.006. Epub 2014 Jul 24.
9
Dengue induces platelet activation, mitochondrial dysfunction and cell death through mechanisms that involve DC-SIGN and caspases.登革热通过涉及 DC-SIGN 和半胱天冬酶的机制诱导血小板活化、线粒体功能障碍和细胞死亡。
J Thromb Haemost. 2013 May;11(5):951-62. doi: 10.1111/jth.12178.
10
Eventual role of asymptomatic cases of dengue for the introduction and spread of dengue viruses in non-endemic regions.登革热无症状病例在登革热病毒传入和传播至非流行地区过程中的最终作用。
Front Physiol. 2012 Mar 30;3:70. doi: 10.3389/fphys.2012.00070. eCollection 2012.

New insight on dengue virus-induced thrombocytopenia.

作者信息

Goldthorpe Stacy C, Conway Michael J

机构信息

a Foundational Sciences , Central Michigan University College of Medicine , Mount Pleasant , MI , USA.

出版信息

Virulence. 2017 Nov 17;8(8):1492-1493. doi: 10.1080/21505594.2017.1368943. Epub 2017 Sep 21.

DOI:10.1080/21505594.2017.1368943
PMID:28829671
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC5810472/
Abstract
摘要