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核心技术专利：CN118964589B侵权必究

核心技术专利：CN118964589B侵权必究

Akoev G N, Andrianov G N, Szabo T

Pavlov Institute of Physiology, Academy of Sciences of the USSR, Leningrad.

Comp Biochem Physiol A Comp Physiol. 1987;88(1):61-3. doi: 10.1016/0300-9629(87)90099-5.

Effects of kainic acid (KA) on resting and evoked activities of the ampullary electroreceptor were studied in marine skates (Raja clavata). 2. Perfusion of the basal membrane with 10(-6)-10(-9) M KA produced significant and reversible changes in impulse activity depending on initial firing rate. 3. When synaptic transmission was blocked by perfusion with elevated Mg2+, the resting and evoked activities were restored if KA was added. 4. The results are consistent with the view that KA is a potent excitant of the ampullae receptors and its effects appear to be presynaptic.

Akoev G N, Andrianov G N, Szabo T

Pavlov Institute of Physiology, Academy of Sciences of the USSR, Leningrad.

Comp Biochem Physiol A Comp Physiol. 1987;88(1):61-3. doi: 10.1016/0300-9629(87)90099-5.

Effects of kainic acid (KA) on resting and evoked activities of the ampullary electroreceptor were studied in marine skates (Raja clavata). 2. Perfusion of the basal membrane with 10(-6)-10(-9) M KA produced significant and reversible changes in impulse activity depending on initial firing rate. 3. When synaptic transmission was blocked by perfusion with elevated Mg2+, the resting and evoked activities were restored if KA was added. 4. The results are consistent with the view that KA is a potent excitant of the ampullae receptors and its effects appear to be presynaptic.