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Renal physiology: Prostaglandins in thiazide-induced hyponatraemia: do they hold water?

作者信息

Hoorn Ewout J, Wetzels Jack F M

机构信息

Department of Internal Medicine, Division of Nephrology &Transplantation, Erasmus Medical Center, Room H-438, PO Box 2040, 3000 CA, Rotterdam, Netherlands.

Department of Nephrology 464, Radboud University Medical Center, PO Box 9101, 6500 HB Nijmegen, Netherlands.

出版信息

Nat Rev Nephrol. 2017 Nov;13(11):665-666. doi: 10.1038/nrneph.2017.133. Epub 2017 Sep 25.

DOI:10.1038/nrneph.2017.133
PMID:28944774
Abstract
摘要

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本文引用的文献

1
Phenotypic and pharmacogenetic evaluation of patients with thiazide-induced hyponatremia.噻嗪类药物所致低钠血症患者的表型和药物遗传学评估
J Clin Invest. 2017 Sep 1;127(9):3367-3374. doi: 10.1172/JCI89812. Epub 2017 Aug 7.
2
Thiazide-induced hyponatraemia is associated with increased water intake and impaired urea-mediated water excretion at low plasma antidiuretic hormone and urine aquaporin-2.噻嗪类药物引起的低钠血症与水摄入量增加以及在低血浆抗利尿激素和尿水通道蛋白-2水平时尿素介导的水排泄受损有关。
J Hypertens. 2015 Mar;33(3):627-33. doi: 10.1097/HJH.0000000000000423.
3
Is there a role for PGE2 in urinary concentration?
PGE2 在尿浓缩中是否起作用?
J Am Soc Nephrol. 2013 Feb;24(2):169-78. doi: 10.1681/ASN.2012020217. Epub 2012 Nov 15.
4
Antidiuretic effect of hydrochlorothiazide in lithium-induced nephrogenic diabetes insipidus is associated with upregulation of aquaporin-2, Na-Cl co-transporter, and epithelial sodium channel.氢氯噻嗪对锂诱导的肾性尿崩症的抗利尿作用与水通道蛋白-2、钠-氯共转运体和上皮钠通道的上调有关。
J Am Soc Nephrol. 2004 Nov;15(11):2836-43. doi: 10.1097/01.ASN.0000143476.93376.04.
5
Risk factors for thiazide-induced hyponatraemia.噻嗪类药物所致低钠血症的危险因素。
QJM. 2003 Dec;96(12):911-7. doi: 10.1093/qjmed/hcg157.
6
Evidence that chronicity of hyponatremia contributes to the high urate clearance observed in the syndrome of inappropriate antidiuretic hormone secretion.低钠血症的慢性状态导致抗利尿激素分泌不当综合征中观察到的高尿酸清除率的证据。
Am J Kidney Dis. 2000 Oct;36(4):745-51. doi: 10.1053/ajkd.2000.17623.
7
Thiazide induces water absorption in the inner medullary collecting duct of normal and Brattleboro rats.噻嗪类药物可诱导正常大鼠和布拉特洛伐大鼠的髓质内集合管对水的重吸收。
Am J Physiol. 1999 Nov;277(5):F756-60. doi: 10.1152/ajprenal.1999.277.5.F756.
8
Increased susceptibility to thiazide-induced hyponatremia in the elderly.老年人对噻嗪类药物所致低钠血症的易感性增加。
J Am Soc Nephrol. 1994 Oct;5(4):1106-11. doi: 10.1681/ASN.V541106.
9
Interaction of bemetizide and indomethacin in the kidney.苄氟噻嗪与吲哚美辛在肾脏中的相互作用。
Br J Clin Pharmacol. 1983 Oct;16(4):377-83. doi: 10.1111/j.1365-2125.1983.tb02181.x.
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Thiazide-induced hyponatremia. Reproducibility by single dose rechallenge and an analysis of pathogenesis.噻嗪类药物所致低钠血症。单次剂量再激发试验的可重复性及发病机制分析
Ann Intern Med. 1989 Jan 1;110(1):24-30. doi: 10.7326/0003-4819-110-1-24.