Asthma predisposition and respiratory syncytial virus infection modulate transient receptor potential vanilloid 1 function in children's airways.
作者信息
Harford Terri J, Rezaee Fariba, Scheraga Rachel G, Olman Mitchell A, Piedimonte Giovanni
机构信息
Center for Pediatric Research, Pediatric Institute and Children's Hospital, The Cleveland Clinic, Cleveland, Ohio.
Department of Pathobiology, Lerner Research Institute, The Cleveland Clinic, Cleveland, Ohio.
出版信息
J Allergy Clin Immunol. 2018 Jan;141(1):414-416.e4. doi: 10.1016/j.jaci.2017.07.015. Epub 2017 Oct 2.
Abstract
摘要
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本文引用的文献
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Respiratory virus infection up-regulates TRPV1, TRPA1 and ASICS3 receptors on airway cells.呼吸道病毒感染会上调气道细胞上的TRPV1、TRPA1和ASIC3受体。
PLoS One. 2017 Feb 10;12(2):e0171681. doi: 10.1371/journal.pone.0171681. eCollection 2017.
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Increased expression of bronchial epithelial transient receptor potential vanilloid 1 channels in patients with severe asthma.在严重哮喘患者的支气管上皮细胞中，瞬时受体电位香草素 1 通道的表达增加。
J Allergy Clin Immunol. 2014 Mar;133(3):704-12.e4. doi: 10.1016/j.jaci.2013.09.016. Epub 2013 Nov 8.
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Transient receptor potential vanilloid 1 receptors mediate acid-induced mucin secretion via Ca2+ influx in human airway epithelial cells.瞬时受体电位香草酸 1 型受体通过钙离子内流介导人呼吸道上皮细胞的酸诱导粘蛋白分泌。
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Bronchoconstriction induced by hyperventilation with humidified hot air: role of TRPV1-expressing airway afferents.加湿热空气过度通气诱导的支气管收缩：表达TRPV1的气道传入神经的作用
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Activation of transient receptor potential A1 channels by mustard oil, tetrahydrocannabinol and Ca2+ reveals different functional channel states.芥子油、四氢大麻酚和Ca2+对瞬时受体电位A1通道的激活揭示了不同的功能性通道状态。
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TRPV1 activation results in disruption of the blood-brain barrier in the rat.瞬时受体电位香草酸亚型1（TRPV1）激活会导致大鼠血脑屏障的破坏。
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Increased expression of transient receptor potential vanilloid-1 in airway nerves of chronic cough.慢性咳嗽患者气道神经中瞬时受体电位香草酸亚型1表达增加。
Am J Respir Crit Care Med. 2004 Dec 15;170(12):1276-80. doi: 10.1164/rccm.200402-174OC. Epub 2004 Sep 24.
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The capsaicin receptor: a heat-activated ion channel in the pain pathway.辣椒素受体：疼痛通路中的一种热激活离子通道。
Nature. 1997 Oct 23;389(6653):816-24. doi: 10.1038/39807.
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