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Transglutaminase-mediated cross-linking of proteins and cell ageing: the erythrocyte and lens models.

作者信息

Lorand L

机构信息

Department of Biochemistry, Molecular Biology and Cell Biology, Northwestern University Evanston, IL 60201.

出版信息

Adv Exp Med Biol. 1988;231:79-94. doi: 10.1007/978-1-4684-9042-8_7.

DOI:10.1007/978-1-4684-9042-8_7
PMID:2901196
Abstract
摘要

相似文献

1
Transglutaminase-mediated cross-linking of proteins and cell ageing: the erythrocyte and lens models.
Adv Exp Med Biol. 1988;231:79-94. doi: 10.1007/978-1-4684-9042-8_7.
2
Post-translational modifications of eye lens crystallins: crosslinking, phosphorylation and deamidation.眼晶状体晶状体蛋白的翻译后修饰:交联、磷酸化和脱酰胺作用。
Adv Exp Med Biol. 1988;231:95-108. doi: 10.1007/978-1-4684-9042-8_8.
3
Properties of purified lens transglutaminase and regulation of its transamidase/crosslinking activity by GTP.纯化晶状体转谷氨酰胺酶的特性及其转氨/交联活性受GTP的调控
Exp Eye Res. 1998 Sep;67(3):273-81. doi: 10.1006/exer.1998.0509.
4
Transglutaminase-mediated cross-linking of alpha-crystallin: structural and functional consequences.转谷氨酰胺酶介导的α-晶状体蛋白交联:结构和功能后果
FEBS Lett. 2001 Jun 22;499(3):245-50. doi: 10.1016/s0014-5793(01)02565-0.
5
Acceptor-donor relationships in the transglutaminase-mediated cross-linking of lens beta-crystallin subunits.转谷氨酰胺酶介导的晶状体β-晶状体蛋白亚基交联中的受体-供体关系。
Biochemistry. 1987 Jul 28;26(15):4629-34. doi: 10.1021/bi00389a006.
6
Spermidine delays eye lens opacification in vitro by suppressing transglutaminase-catalyzed crystallin cross-linking.亚精胺通过抑制转谷氨酰胺酶催化的晶状体蛋白交联来延缓体外晶状体混浊。
Protein J. 2011 Feb;30(2):109-14. doi: 10.1007/s10930-011-9311-7.
7
Cross-linking and proteolysis in Ca2(+)-treated lens homogenates.钙离子处理的晶状体匀浆中的交联和蛋白水解作用。
Biochim Biophys Acta. 1990 Sep 3;1040(2):187-91. doi: 10.1016/0167-4838(90)90074-p.
8
Novel inhibitors against the transglutaminase-catalysed crosslinking of lens proteins.新型抗转谷氨酰胺酶催化晶状体蛋白交联的抑制剂
Exp Eye Res. 1998 May;66(5):531-6. doi: 10.1006/exer.1997.0463.
9
Long-term organ culture system to study the effects of UV-A irradiation on lens transglutaminase.
Metab Pediatr Syst Ophthalmol (1985). 1996;19-20:23-6.
10
Inhibition of beta-crystallin cross-linking in the Ca2+-treated lens.在经钙离子处理的晶状体中β-晶状体蛋白交联的抑制作用
Invest Ophthalmol Vis Sci. 1987 Jul;28(7):1218-22.

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Thiol-Mediated Enhancement of N-Acetyllysine Formation in Lens Proteins.巯基介导的晶状体蛋白 N-乙酰赖氨酸形成的增强作用。
ACS Chem Biol. 2024 Jul 19;19(7):1495-1505. doi: 10.1021/acschembio.4c00174. Epub 2024 Jun 21.
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Identification of crystallin modifications in the human lens cortex and nucleus using laser capture microdissection and CyDye labeling.使用激光捕获显微切割和CyDye标记技术鉴定人晶状体皮质和核中的晶状体蛋白修饰。
Mol Vis. 2010 Mar 23;16:476-94.
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UV-A-induced structural and functional changes in human lens deamidated alphaB-crystallin.
紫外线A诱导人晶状体脱酰胺αB-晶状体蛋白的结构和功能变化。
Mol Vis. 2008 Feb 1;14:234-48.
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Protein kinase catalytic subunit (PKAcat) from bovine lens: purification, characterization and phosphorylation of lens crystallins.
Mol Cell Biochem. 2007 Oct;304(1-2):155-65. doi: 10.1007/s11010-007-9496-4. Epub 2007 May 25.
5
Induction of tissue transglutaminase by dexamethasone: its correlation to receptor number and transglutaminase-mediated cell death in a series of malignant hamster fibrosarcomas.地塞米松对组织转谷氨酰胺酶的诱导作用:在一系列恶性仓鼠纤维肉瘤中其与受体数量及转谷氨酰胺酶介导的细胞死亡的相关性
Biochem J. 1998 Apr 1;331 ( Pt 1)(Pt 1):105-12. doi: 10.1042/bj3310105.
6
Transglutaminase induction by various cell death and apoptosis pathways.通过各种细胞死亡和凋亡途径诱导转谷氨酰胺酶
Experientia. 1996 Oct 31;52(10-11):942-9. doi: 10.1007/BF01920102.