Gibbons R J, Hay D I, Cisar J O, Clark W B
Forsyth Dental Center, Boston, Massachusetts 02115.
Infect Immun. 1988 Nov;56(11):2990-3. doi: 10.1128/iai.56.11.2990-2993.1988.
Use of specific fimbria-defective mutants derived from Actinomyces viscosus T14 has shown that salivary acidic proline-rich protein 1 and statherin serve as receptors for type 1 fimbriae which mediate attachment of the organism to experimental pellicles on apatitic surfaces.
源自粘性放线菌T14的特定菌毛缺陷突变体的使用表明,唾液酸性富含脯氨酸蛋白1和磷蛋白作为1型菌毛的受体,介导该生物体附着于磷灰石表面的实验性薄膜。