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Response to letter to the editor of Carcinogenesis by Pira et al., 2017.对皮拉等人于2017年致《癌变》杂志编辑信件的回复。
Carcinogenesis. 2017 Dec 7;38(12):1253-1255. doi: 10.1093/carcin/bgx111.
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Hematologic and cytogenetic biomarkers of leukemia risk from formaldehyde exposure.甲醛暴露导致白血病风险的血液学和细胞遗传学生物标志物。
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Response letter to the editor RE: Formaldehyde and leukemia: missing evidence!致编辑的回信 主题：甲醛与白血病：证据缺失！
Cancer Causes Control. 2013 Jan;24(1):205. doi: 10.1007/s10552-012-0112-x. Epub 2012 Nov 29.
4
Does occupational exposure to formaldehyde cause hematotoxicity and leukemia-specific chromosome changes in cultured myeloid progenitor cells?职业性接触甲醛是否会导致培养的髓系祖细胞的血液毒性和白血病特异性染色体改变？
Crit Rev Toxicol. 2017 Aug;47(7):592-602. doi: 10.1080/10408444.2017.1301878. Epub 2017 May 2.
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Hypersensitivity to formaldehyde.对甲醛过敏。
Allergy. 2000 Oct;55(10):986-7. doi: 10.1034/j.1398-9995.2000.00814.x.
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Using mechanistic information to support evidence integration and synthesis: a case study with inhaled formaldehyde and leukemia.利用机制信息支持证据的综合与整合：以吸入甲醛与白血病为例。
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7
Is it possible to use biomonitoring for the quantitative assessment of formaldehyde occupational exposure?是否可以使用生物监测来对甲醛职业暴露进行定量评估？
Biomark Med. 2016 Dec;10(12):1287-1303. doi: 10.2217/bmm-2016-0146. Epub 2016 Dec 7.
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Indoor exposure to formaldehyde and risk of allergy.室内接触甲醛与过敏风险
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Is formaldehyde an important cause of allergic respiratory disease?甲醛是过敏性呼吸道疾病的重要病因吗？
Clin Exp Allergy. 1996 Mar;26(3):247-9.
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Formaldehyde reactions and the burden of proof.甲醛反应与举证责任。
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引用本文的文献

1
Formaldehyde, Hematotoxicity, and Chromosomal Changes-Response.甲醛、血液毒性与染色体变化——反应
Cancer Epidemiol Biomarkers Prev. 2018 Jan;27(1):120-121. doi: 10.1158/1055-9965.EPI-17-0804.

本文引用的文献

1
Hematologic and cytogenetic biomarkers of leukemia risk from formaldehyde exposure.甲醛暴露导致白血病风险的血液学和细胞遗传学生物标志物。
Carcinogenesis. 2017 Dec 7;38(12):1251-1252. doi: 10.1093/carcin/bgx072.
2
Mutational Landscape and Gene Expression Patterns in Adult Acute Myeloid Leukemias with Monosomy 7 as a Sole Abnormality.以7号染色体单体作为唯一异常的成人急性髓系白血病的突变图谱和基因表达模式
Cancer Res. 2017 Jan 1;77(1):207-218. doi: 10.1158/0008-5472.CAN-16-1386. Epub 2016 Oct 26.
3
Chromosome-wide aneuploidy study of cultured circulating myeloid progenitor cells from workers occupationally exposed to formaldehyde.职业性接触甲醛工人培养循环髓系祖细胞的全染色体非整倍体研究。
Carcinogenesis. 2015 Jan;36(1):160-7. doi: 10.1093/carcin/bgu229. Epub 2014 Nov 12.
4
Chemical agents and related occupations.化学制剂及相关职业。
IARC Monogr Eval Carcinog Risks Hum. 2012;100(Pt F):9-562.
5
Leukemia-related chromosomal loss detected in hematopoietic progenitor cells of benzene-exposed workers.苯暴露工人造血祖细胞中检测到与白血病相关的染色体缺失。
Leukemia. 2012 Dec;26(12):2494-8. doi: 10.1038/leu.2012.143. Epub 2012 May 30.
6
Chromosomics: detection of numerical and structural alterations in all 24 human chromosomes simultaneously using a novel OctoChrome FISH assay.染色体组学：使用一种新型八色荧光原位杂交（OctoChrome FISH）检测法同时检测人类所有24条染色体的数目和结构改变。
J Vis Exp. 2012 Feb 6(60):3619. doi: 10.3791/3619.
7
Chromosome-wide aneuploidy study (CWAS) in workers exposed to an established leukemogen, benzene.染色体-wide 非整倍性研究 (CWAS) 在工人接触到既定的白血病发生物，苯。
Carcinogenesis. 2011 Apr;32(4):605-12. doi: 10.1093/carcin/bgq286. Epub 2011 Jan 7.
8
Occupational exposure to formaldehyde, hematotoxicity, and leukemia-specific chromosome changes in cultured myeloid progenitor cells.职业性接触甲醛、血液毒性与培养的髓性造血祖细胞白血病特异性染色体改变
Cancer Epidemiol Biomarkers Prev. 2010 Jan;19(1):80-8. doi: 10.1158/1055-9965.EPI-09-0762.
9
Use of OctoChrome fluorescence in situ hybridization to detect specific aneuploidy among all 24 chromosomes in benzene-exposed workers.使用八色荧光原位杂交技术检测苯接触工人所有24条染色体中的特定非整倍体。
Chem Biol Interact. 2005 May 30;153-154:117-22. doi: 10.1016/j.cbi.2005.03.016. Epub 2005 Apr 19.
10
The nature of chromosomal aberrations detected in humans exposed to benzene.在接触苯的人群中检测到的染色体畸变的性质。
Crit Rev Toxicol. 2002 Jan;32(1):1-42. doi: 10.1080/20024091064165.

Response to letter to the editor of Carcinogenesis by Pira et al., 2017.

作者信息

Rothman Nathaniel, Lan Qing, Smith Martyn T, Vermeulen Roel, Zhang Luoping

机构信息

Occupational and Environmental Epidemiology Branch, Division of Cancer, Epidemiology and Genetics, Department of Health and Human Services, National Cancer Institute (NIH), USA.

Division of Environmental Health Sciences, School of Public Health, University of California at Berkeley, USA.

出版信息

Carcinogenesis. 2017 Dec 7;38(12):1253-1255. doi: 10.1093/carcin/bgx111.

DOI:10.1093/carcin/bgx111
PMID:29040449
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC5862264/
Abstract
摘要