Kul'chitskiĭ V A, Tkachenko B I
Fiziol Zh SSSR Im I M Sechenova. 1988 Oct;74(10):1409-17.
In acute experiments on cats, under constant perfusion of tibial and small intestine vessels and after bilateral electrolytic lesion of ventral portions of the paragigantocellular nucleus a decrease of initial AP by 30-35%, perfusion pressure in tibial vessels by 30% was observed whereas the changes of AP in the small intestine vessel were insignificant. The occlusion of the common carotid artery or electrical stimulation of afferent fibers of brachial plexus exerted no effect, at that. Bilateral administration of L-glutamate into ventral portions of reticular nucleus increased the perfusion pressure in tibial vessels by 15% and in those of small intestine by 35%. Carotid sinus pressor reflex induced 1.5-fold shifts of systemic AP as compared with control values.
在对猫进行的急性实验中，在持续灌注胫部和小肠血管的情况下，以及在对巨细胞旁核腹侧部分进行双侧电解损伤后，观察到初始动作电位降低了30 - 35%，胫部血管的灌注压力降低了30%，而小肠血管中动作电位的变化不显著。此时，阻断颈总动脉或电刺激臂丛神经的传入纤维均无作用。向网状核腹侧部分双侧注射L - 谷氨酸，可使胫部血管的灌注压力升高15%，小肠血管的灌注压力升高35%。与对照值相比，颈动脉窦升压反射使全身动作电位发生了1.5倍的变化。
