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TRPA1 channel participates in tacrolimus-induced pruritus in a chronic contact hypersensitivity murine model.

作者信息

Wong Lai San, Otsuka Atsushi, Yamamoto Yasuo, Nonomura Yumi, Nakashima Chisa, Kitayama Naomi, Usui Kenji, Honda Tetsuya, Kabashima Kenji

机构信息

Department of Dermatology, Kyoto University Graduate School of Medicine, Kyoto, Japan; Department of Dermatology, Kaohsiung Chang Gung Memorial Hospital and Chang Gung University College of Medicine, Kaohsiung, Taiwan.

Department of Dermatology, Kyoto University Graduate School of Medicine, Kyoto, Japan; Translational research department for skin and brain diseases, Kyoto University Graduate School of Medicine, 54 Shogoin-Kawara, Sakyo-ku, Kyoto, Japan.

出版信息

J Dermatol Sci. 2018 Feb;89(2):207-209. doi: 10.1016/j.jdermsci.2017.10.012. Epub 2017 Oct 31.

DOI:10.1016/j.jdermsci.2017.10.012
PMID:29128286
Abstract
摘要

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引用本文的文献

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Targeting Transient Receptor Potential (TRP) Channels, Mas-Related G-Protein-Coupled Receptors (Mrgprs), and Protease-Activated Receptors (PARs) to Relieve Itch.靶向瞬时受体电位(TRP)通道、Mas相关G蛋白偶联受体(Mrgprs)和蛋白酶激活受体(PARs)以缓解瘙痒。
Pharmaceuticals (Basel). 2023 Dec 8;16(12):1707. doi: 10.3390/ph16121707.
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The Role of Transient Receptor Potential A1 and G Protein-Coupled Receptor 39 in Zinc-Mediated Acute and Chronic Itch in Mice.
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