Duangthip Duangporn, Man Arthur, Poon Pak Hong, Lo Edward Chin Man, Chu Chun-Hung
Faculty of Dentistry, The University of Hong Kong, Hong Kong, China.
Am J Dent. 2017 Aug;30(4):212-220.
This systematic review on abfraction studied whether stress is a mechanism in the formation of non-carious cervical lesions (NCCLs).
A literature search was performed on three electronic databases (PubMed, ISI Web of Science, and EMBASE) using the keyword "abfraction" in publications published in English. The inclusion criteria were clinical and laboratory studies that investigated the role of abfraction in NCCLs. The title and abstract of the identified publications were screened by two investigators independently. Reviews, case reports, and irrelevant papers were excluded. Full text of the remaining publications were retrieved. A manual search was performed on the bibliographies of the selected publications to identify additional relevant publications for review.
A total of 372 publications were identified, and 165 duplicated publications and 166 irrelevant publications were excluded. From the bibliographies of the remaining 41 publications, 28 relevant publications were found. Therefore, 69 publications (31 clinical studies and 38 laboratory studies) were included in this review and the majority (56/69, 81%) found an association between occlusal stress and NCCLs. Although no clinical study demonstrated that NCCL was caused by stress alone, 23 studies reported that stress or occlusal factors were associated with NCCLs. Of the 38 laboratory studies, 24 that used finite element analysis found that stress was concentrated at the cervical region of the tooth. Nine laboratory studies suggested that stress was a mechanism for NCCLs, whereas five studies reported the opposite. In conclusion, current literature supported an association between occlusal stress and NCCLs.
This systematic review of abfraction found the majority of studies reported an association between occlusal stress and non-carious cervical lesions.
本项关于磨损的系统评价研究了应力是否为非龋性颈部病变(NCCLs)形成的一种机制。
在三个电子数据库(PubMed、ISI 科学网和 EMBASE)中进行文献检索,使用英文发表的文献中的关键词“磨损”。纳入标准为调查磨损在 NCCLs 中作用的临床和实验室研究。两名研究者独立筛选所识别文献的标题和摘要。综述、病例报告及无关论文被排除。检索其余文献的全文。对所选文献的参考文献进行手工检索,以识别其他相关文献进行综述。
共识别出372篇文献,排除165篇重复文献和166篇无关文献。从其余41篇文献的参考文献中,发现28篇相关文献。因此,本综述纳入69篇文献(31篇临床研究和38篇实验室研究),大多数(56/69,81%)发现咬合应力与NCCLs之间存在关联。虽然没有临床研究表明NCCL仅由应力引起,但23项研究报告应力或咬合因素与NCCLs相关。在38项实验室研究中,24项使用有限元分析的研究发现应力集中在牙齿颈部区域。9项实验室研究表明应力是NCCLs的一种机制,而5项研究报告结果相反。总之,当前文献支持咬合应力与NCCLs之间存在关联。
本项关于磨损的系统评价发现,大多数研究报告咬合应力与非龋性颈部病变之间存在关联。