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Vitamin D metabolism in tumoral calcinosis.

作者信息

Steinherz R

出版信息

Eur J Pediatr. 1989 Feb;148(5):475. doi: 10.1007/BF00595920.

DOI:10.1007/BF00595920
PMID:2920757
Abstract
摘要

相似文献

1
Vitamin D metabolism in tumoral calcinosis.
Eur J Pediatr. 1989 Feb;148(5):475. doi: 10.1007/BF00595920.
2
Hyperphosphatemic tumoral calcinosis: effects of phosphate depletion on vitamin D metabolism, and of acute hypocalcemia on parathyroid hormone secretion and action.高磷血症性肿瘤性钙化：磷缺乏对维生素D代谢的影响，以及急性低钙血症对甲状旁腺激素分泌和作用的影响。
J Clin Endocrinol Metab. 1983 Jun;56(6):1319-22. doi: 10.1210/jcem-56-6-1319.
3
Massive tumoral calcinosis in a patient on long-term hemodialysis.
J Bone Miner Res. 2000 Oct;15(10):2056-7. doi: 10.1359/jbmr.2000.15.10.2056.
4
Vitamin D metabolites in generalized scleroderma. Evidence of a normal cutaneous and intestinal supply with vitamin D.系统性硬皮病中的维生素D代谢产物。维生素D皮肤和肠道正常供应的证据。
Acta Derm Venereol. 1985;65(4):343-5.
5
FGF-23, vitamin D and calcification: the unholy triad.成纤维细胞生长因子-23、维生素D与钙化：邪恶三联征
Nephrol Dial Transplant. 2005 Oct;20(10):2032-5. doi: 10.1093/ndt/gfh991. Epub 2005 Jul 19.
6
A homozygous missense mutation in human KLOTHO causes severe tumoral calcinosis.人类KLOTHO基因的纯合错义突变会导致严重的肿瘤性钙化。
J Musculoskelet Neuronal Interact. 2007 Oct-Dec;7(4):318-9.
7
A novel mutation in fibroblast growth factor 23 gene as a cause of tumoral calcinosis.成纤维细胞生长因子23基因的一种新突变作为肿瘤性钙化的病因
J Clin Endocrinol Metab. 2005 Oct;90(10):5523-7. doi: 10.1210/jc.2005-0301. Epub 2005 Jul 19.
8
Phosphorus excretion in tumoral calcinosis: response to parathyroid hormone and acetazolamide.
J Clin Endocrinol Metab. 1980 Apr;50(4):648-53. doi: 10.1210/jcem-50-4-648.
9
Hyperphosphatemic familial tumoral calcinosis: genetic models of deficient FGF23 action.高磷血症性家族性肿瘤性钙化症：FGF23作用缺陷的遗传模型
Curr Osteoporos Rep. 2015 Apr;13(2):78-87. doi: 10.1007/s11914-015-0254-3.
10
Calcium and phosphate metabolism in tumoral calcinosis.
Ann Intern Med. 1980 Apr;92(4):482-7. doi: 10.7326/0003-4819-92-4-482.

本文引用的文献

1
Hyperphosphatemic tumoral calcinosis: association with elevation of serum 1,25-dihydroxycholecalciferol concentrations.高磷血症性肿瘤性钙化：与血清1,25-二羟胆钙化醇浓度升高相关。
Ann Intern Med. 1982 May;96(5):586-91. doi: 10.7326/0003-4819-96-5-586.
2
Tumoral calcinosis: evidence for concurrent defects in renal tubular phosphorus transport and in 1 alpha,25-dihydroxycholecalciferol synthesis.肿瘤性钙化：肾小管磷转运及1α,25-二羟胆钙化醇合成同时存在缺陷的证据。
Calcif Tissue Int. 1980;32(1):1-6. doi: 10.1007/BF02408515.
3
Hyperphosphatemic tumoral calcinosis: effects of phosphate depletion on vitamin D metabolism, and of acute hypocalcemia on parathyroid hormone secretion and action.
高磷血症性肿瘤性钙化：磷缺乏对维生素D代谢的影响，以及急性低钙血症对甲状旁腺激素分泌和作用的影响。
J Clin Endocrinol Metab. 1983 Jun;56(6):1319-22. doi: 10.1210/jcem-56-6-1319.
4
Elevated serum calcitriol concentrations do not fall in response to hyperphosphatemia in familial tumoral calcinosis.在家族性肿瘤性钙化症中，血清骨化三醇浓度升高不会因高磷血症而下降。
Am J Dis Child. 1985 Aug;139(8):816-9. doi: 10.1001/archpedi.1985.02140100078036.
5
Tumoral calcinosis revisited--common and uncommon features. Report of ten cases and review.肿瘤性钙化症再探讨——常见与不常见特征。十例报告及文献复习
Eur J Pediatr. 1988 Feb;147(2):128-32. doi: 10.1007/BF00442209.
6
The importance of phosphate in regulating plasma 1,25-(OH)2-vitamin D levels in humans: studies in healthy subjects in calcium-stone formers and in patients with primary hyperparathyroidism.
J Clin Endocrinol Metab. 1977 Aug;45(2):299-306. doi: 10.1210/jcem-45-2-299.