Normal plasma IgG inhibits HIT antibody-mediated platelet activation: implications for therapeutic plasma exchange.
作者信息
Jones Curtis G, Pechauer Shannon M, Curtis Brian R, Bougie Daniel W, Aster Richard H, Padmanabhan Anand
机构信息
Medical Sciences Institute.
Blood Research Institute, and.
出版信息
Blood. 2018 Feb 8;131(6):703-706. doi: 10.1182/blood-2017-08-803031. Epub 2017 Dec 19.
Abstract
摘要
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Normal plasma IgG inhibits HIT antibody-mediated platelet activation: implications for therapeutic plasma exchange.正常血浆IgG可抑制肝素诱导的血小板减少症（HIT）抗体介导的血小板活化：对治疗性血浆置换的意义。
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IVIg for Treatment of Severe Refractory Heparin-Induced Thrombocytopenia.静脉注射免疫球蛋白治疗严重难治性肝素诱导的血小板减少症
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3
Clinical outcomes in a cohort of patients with heparin-induced thrombocytopenia.肝素诱导的血小板减少症患者队列的临床结局。
Am J Hematol. 2017 Aug;92(8):730-738. doi: 10.1002/ajh.24759. Epub 2017 Apr 26.
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Guidelines on the Use of Therapeutic Apheresis in Clinical Practice-Evidence-Based Approach from the Writing Committee of the American Society for Apheresis: The Seventh Special Issue.《临床实践中治疗性血液成分单采的使用指南——美国血液成分单采协会写作委员会基于证据的方法：第七期特刊》
J Clin Apher. 2016 Jun;31(3):149-62. doi: 10.1002/jca.21470.
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Transfusion Reactions.输血反应
Hematol Oncol Clin North Am. 2016 Jun;30(3):619-34. doi: 10.1016/j.hoc.2016.01.012.
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CLINICAL PRACTICE. Heparin-Induced Thrombocytopenia.临床实践。肝素诱导的血小板减少症。
N Engl J Med. 2015 Jul 16;373(3):252-61. doi: 10.1056/NEJMcp1411910.
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Severe and persistent heparin-induced thrombocytopenia despite fondaparinux treatment.尽管使用磺达肝癸钠治疗，仍出现严重且持续性肝素诱导的血小板减少症。
Am J Hematol. 2015 Jul;90(7):675-8. doi: 10.1002/ajh.23971. Epub 2015 Feb 27.
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Increased risk of thrombosis in FcγRIIA 131RR patients with HIT due to defective control of platelet activation by plasma IgG2.由于血浆 IgG2 对血小板活化的控制缺陷，导致 HIT 患者 FcγRIIA 131RR 发生血栓形成风险增加。
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9
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Advances in the pathophysiology and treatment of heparin-induced thrombocytopenia.肝素诱导的血小板减少症的病理生理学与治疗进展
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