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Auditory agnosia caused by bilateral putamen haemorrhage.双侧壳核出血所致听觉失认症。
BMJ Case Rep. 2017 Dec 20;2017:bcr-2017-222535. doi: 10.1136/bcr-2017-222535.
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Subcortical deafness as a subtype of auditory agnosia after injury of bilateral auditory radiations caused by two cerebrovascular accidents - normal auditory brainstem responses with I-VII waves and abolished consciousness of hearing.双侧听觉辐射损伤导致的两次脑血管意外后的皮质下聋作为听觉失认症的一个亚型——听觉脑干反应 I-VII 波正常,听觉意识丧失。
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本文引用的文献

1
What is involved and what is necessary for complex linguistic and nonlinguistic auditory processing: evidence from functional magnetic resonance imaging and lesion data.复杂语言和非语言听觉处理涉及哪些方面以及需要什么:来自功能磁共振成像和病变数据的证据。
J Cogn Neurosci. 2007 May;19(5):799-816. doi: 10.1162/jocn.2007.19.5.799.
2
Magnetoencephalography and positron emission tomography studies of a patient with auditory agnosia caused by bilateral lesions confined to the auditory radiations.
Acta Otolaryngol. 2005 Dec;125(12):1351-5. doi: 10.1080/00016480510027501.
3
Cortical auditory disorder caused by bilateral temporal infarctions.
Intern Med. 1995 Aug;34(8):801-5. doi: 10.2169/internalmedicine.34.801.
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Cortical deafness--a case report and review of the literature.
J Neurol Sci. 1980 Oct;48(1):35-49. doi: 10.1016/0022-510x(80)90148-3.
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'So-called' cortical deafness. Clinical, neurophysiological and radiological observations.“所谓的”皮质性聋。临床、神经生理学及放射学观察
Brain. 1991 Dec;114 ( Pt 6):2385-401. doi: 10.1093/brain/114.6.2385.

双侧壳核出血所致听觉失认症。

Auditory agnosia caused by bilateral putamen haemorrhage.

作者信息

Sugiura Tomohito, Torii Tsuyoshi

机构信息

Department of Neurology, National Hospital Organization Kure Medical Center, 3-1 Aoyama, Kure city, Japan.

出版信息

BMJ Case Rep. 2017 Dec 20;2017:bcr-2017-222535. doi: 10.1136/bcr-2017-222535.

DOI:10.1136/bcr-2017-222535
PMID:29269367
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC5743879/
Abstract

A 55-year-old right-handed man with a history of hypertension suddenly fell and developed right hemiparesis. Neurological examination revealed that he was alert, but did not appropriately respond to verbal questions and commands. Detailed examination revealed that he could correctly respond to written commands. His speech was almost fluent, showing no paraphasia and normal articulation. His written sentences were legible. Pure tone audiometry showed that his auditory acuity was relatively preserved. His brainstem auditory evoked potential components from I to V were recorded bilaterally with normal latency. Cerebral CT demonstrated fresh bleeding in the left putamen and an old haemorrhage on the opposite side. He was treated by antihypertensive therapy and rehabilitation. Although there remained mild sensory deficit on his right extremities and he felt a slight noise during conversation, he had little difficulty with verbal communication when he was transferred to another hospital on day 38.

摘要

一名55岁的右利手男性,有高血压病史,突然跌倒并出现右侧偏瘫。神经系统检查发现他意识清醒,但对言语问题和指令反应不当。详细检查发现他能正确回应书面指令。他的言语几乎流利,无错语,发音正常。他写的句子清晰可读。纯音听力测试显示他的听力相对保留。双侧记录了他从I到V的脑干听觉诱发电位成分,潜伏期正常。脑部CT显示左侧壳核有新鲜出血,对侧有陈旧性出血。他接受了降压治疗和康复治疗。尽管他的右侧肢体仍有轻度感觉障碍,且在交谈时感到有轻微噪音,但在第38天转至另一家医院时,他在言语交流方面几乎没有困难。