Bayés de Luna A, Carrió I, Subirana M T, Torner P, Cosín J, Sagués F, Guindo J
J Electrocardiol. 1987 Jan;20(1):38-44. doi: 10.1016/0022-0736(87)90006-9.
We studied three groups of individuals by means of spatial-velocity electrocardiograms and thallium-201 myocardial imaging to figure out the electrophysiological explanation of the SI SII SIII electrocardiographic morphology. We studied twelve healthy individuals without SI SII SIII, seven healthy individuals with SI SII SIII and fifteen patients with chronic obstructive pulmonary disease with SI SII SIII. The average values of the QRS-E and QRS-F intervals were higher in the second (P less than 0.05 and P less than 0.005) and third groups (P less than 0.01 and P less than 0.001) than in the first. One patient of the second group and thirteen of the third showed right ventricular enlargement. The slowing down of the right ventricular conduction explained the SI SII SIII morphology in normal individuals in more than half the cases. In patients with chronic obstructive pulmonary disease with SI SII SIII the conduction delay plays an important part in the electrogenesis of the right ventricular enlargement electrocardiographic morphology. We think that these observations can give further data about the electrophysiologic mechanism of the SI SII SIII morphology.
我们通过空间速度心电图和铊-201心肌显像研究了三组个体，以找出SI SII SIII心电图形态的电生理解释。我们研究了12名无SI SII SIII的健康个体、7名有SI SII SIII的健康个体以及15名患有慢性阻塞性肺疾病且有SI SII SIII的患者。第二组（P<0.05和P<0.005）和第三组（P<0.01和P<0.001）的QRS-E和QRS-F间期平均值高于第一组。第二组中有1名患者和第三组中有13名患者出现右心室扩大。在超过半数的正常个体中，右心室传导减慢解释了SI SII SIII形态。在患有慢性阻塞性肺疾病且有SI SII SIII的患者中，传导延迟在右心室扩大心电图形态的电发生中起重要作用。我们认为这些观察结果可以提供有关SI SII SIII形态电生理机制的更多数据。
