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Cross-talk between androgen receptor and nerve growth factor receptor in prostate cancer cells: implications for a new therapeutic approach.

作者信息

Di Donato Marzia, Cernera Gustavo, Auricchio Ferdinando, Migliaccio Antimo, Castoria Gabriella

机构信息

Department of Biochemistry, Biophysics and General Pathology, University of Campania 'Luigi Vanvitelli', Naples, Italy.

出版信息

Cell Death Discov. 2018 Jan 31;4:5. doi: 10.1038/s41420-017-0024-3. eCollection 2018 Dec.

DOI:10.1038/s41420-017-0024-3
PMID:29531802
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC5841355/
Abstract
摘要

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本文引用的文献

1
Extranuclear partners of androgen receptor: at the crossroads of proliferation, migration, and neuritogenesis.雄激素受体的核外伴侣:处于增殖、迁移和神经突形成的交叉点
FASEB J. 2017 Apr;31(4):1289-1300. doi: 10.1096/fj.201601047R. Epub 2016 Dec 28.
2
Cross-talk between androgen receptor/filamin A and TrkA regulates neurite outgrowth in PC12 cells.雄激素受体/细丝蛋白A与酪氨酸激酶受体A之间的相互作用调节PC12细胞中的神经突生长。
Mol Biol Cell. 2015 Aug 1;26(15):2858-72. doi: 10.1091/mbc.E14-09-1352. Epub 2015 Jun 10.
3
Role of non-genomic androgen signalling in suppressing proliferation of fibroblasts and fibrosarcoma cells.非基因组雄激素信号在抑制成纤维细胞和纤维肉瘤细胞增殖中的作用。
Cell Death Dis. 2014 Dec 4;5(12):e1548. doi: 10.1038/cddis.2014.497.
4
Androgen-induced cell migration: role of androgen receptor/filamin A association.雄激素诱导的细胞迁移:雄激素受体/细丝蛋白 A 关联的作用。
PLoS One. 2011 Feb 16;6(2):e17218. doi: 10.1371/journal.pone.0017218.
5
Cytosolic accumulation of gammaH2AX is associated with tropomyosin-related kinase A-induced cell death in U2OS cells.γH2AX在胞质中的积累与原肌球蛋白相关激酶A诱导的U2OS细胞死亡有关。
Exp Mol Med. 2008 Jun 30;40(3):276-85. doi: 10.3858/emm.2008.40.3.276.
6
The dark side of the NGF family: neurotrophins in neoplasias.NGF家族的阴暗面:肿瘤中的神经营养因子
Brain Pathol. 2006 Oct;16(4):304-10. doi: 10.1111/j.1750-3639.2006.00037.x.
7
Mitogenic effect of nerve growth factor (NGF) in LNCaP prostate adenocarcinoma cells: role of the high- and low-affinity NGF receptors.神经生长因子(NGF)对LNCaP前列腺腺癌细胞的促有丝分裂作用:高亲和力和低亲和力NGF受体的作用
Mol Endocrinol. 2000 Jan;14(1):124-36. doi: 10.1210/mend.14.1.0402.