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应激诱导的垂体β-内啡肽释放可能由脑干防御区的激活介导。

Stress-induced release of pituitary beta-endorphin may be mediated by activation of the brain stem defence areas.

作者信息

Haynes L W, Timms R J

出版信息

Int J Tissue React. 1987;9(1):55-9.

PMID:2954924
Abstract

In anaesthetized cats, electrical stimulation within the fore-brain defence areas evoked a marked increase in the plasma level of immunoreactive beta-endorphin which reached a peak after 5 minutes. Multiple immunoreactive peptides including "biologically-active" beta-endorphin-(1-31) were detected. It is suggested that the release of pituitary beta-endorphin during emotional stress results from the natural activation of the defence areas.

摘要

在麻醉猫中,刺激前脑防御区域会引起免疫反应性β-内啡肽血浆水平显著升高,5分钟后达到峰值。检测到多种免疫反应性肽,包括“生物活性”β-内啡肽-(1-31)。有人提出,情绪应激期间垂体β-内啡肽的释放是防御区域自然激活的结果。

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Stress-induced release of pituitary beta-endorphin may be mediated by activation of the brain stem defence areas.应激诱导的垂体β-内啡肽释放可能由脑干防御区的激活介导。
Int J Tissue React. 1987;9(1):55-9.
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