氧自由基在免疫复合物损伤中的作用。
The role of oxygen radicals in immune complex injury.
作者信息
Oldham K T, Guice K S, Ward P A, Johnson K J
机构信息
Department of Surgery, University of Michigan, Ann Arbor 48109.
出版信息
Free Radic Biol Med. 1988;4(6):387-97. doi: 10.1016/0891-5849(88)90090-1.
PMID:2968295
Abstract
In this review we will summarize our current understanding of the mediation of immune complex induced tissue injury. Comparisons will be made between the mediation of IgG versus IgA immune complex injury with emphasis on the role that reactive oxygen products derived from leukocytic phagocytic cells play in the initiation of the tissue injury.
摘要
在本综述中，我们将总结目前对免疫复合物诱导组织损伤介导机制的理解。我们将比较IgG和IgA免疫复合物损伤的介导机制，重点关注白细胞吞噬细胞产生的活性氧产物在组织损伤起始过程中所起的作用。
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