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Correction: Microsomal prostaglandin E synthase-1 gene deletion impairs neuro-immune circuitry of the cholinergic anti-inflammatory pathway in endotoxaemic mouse spleen.更正：微粒体前列腺素E合酶-1基因缺失损害内毒素血症小鼠脾脏中胆碱能抗炎途径的神经免疫回路。
PLoS One. 2018 Apr 26;13(4):e0196806. doi: 10.1371/journal.pone.0196806. eCollection 2018.
2
Microsomal prostaglandin E synthase-1 gene deletion impairs neuro-immune circuitry of the cholinergic anti-inflammatory pathway in endotoxaemic mouse spleen.微粒体前列腺素 E 合酶-1 基因缺失损害内毒素血症小鼠脾胆碱能抗炎途径的神经免疫回路。
PLoS One. 2018 Feb 22;13(2):e0193210. doi: 10.1371/journal.pone.0193210. eCollection 2018.
3
Impaired vagus-mediated immunosuppression in microsomal prostaglandin E synthase-1 deficient mice.微粒体前列腺素E合酶-1缺陷小鼠中迷走神经介导的免疫抑制受损。
Prostaglandins Other Lipid Mediat. 2015 Sep;121(Pt B):155-62. doi: 10.1016/j.prostaglandins.2015.05.006. Epub 2015 May 19.
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引用本文的文献

1
Monocytes Undergo Functional Reprogramming to Generate Immunosuppression through HIF-1 Signaling Pathway in the Late Phase of Sepsis.在脓毒症后期，通过 HIF-1 信号通路，单核细胞经历功能重编程以产生免疫抑制。
Mediators Inflamm. 2020 Feb 7;2020:4235909. doi: 10.1155/2020/4235909. eCollection 2020.

本文引用的文献

1
Microsomal prostaglandin E synthase-1 gene deletion impairs neuro-immune circuitry of the cholinergic anti-inflammatory pathway in endotoxaemic mouse spleen.微粒体前列腺素 E 合酶-1 基因缺失损害内毒素血症小鼠脾胆碱能抗炎途径的神经免疫回路。
PLoS One. 2018 Feb 22;13(2):e0193210. doi: 10.1371/journal.pone.0193210. eCollection 2018.

更正：微粒体前列腺素E合酶-1基因缺失损害内毒素血症小鼠脾脏中胆碱能抗炎途径的神经免疫回路。

Correction: Microsomal prostaglandin E synthase-1 gene deletion impairs neuro-immune circuitry of the cholinergic anti-inflammatory pathway in endotoxaemic mouse spleen.

作者信息

Revathikumar Priya, Estelius Johanna, Karmakar Utsa, Le Maître Erwan, Korotkova Marina, Jakobsson Per-Johan, Lampa Jon

出版信息

PLoS One. 2018 Apr 26;13(4):e0196806. doi: 10.1371/journal.pone.0196806. eCollection 2018.

DOI:10.1371/journal.pone.0196806
PMID:29698528
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC5919627/
Abstract

[This corrects the article DOI: 10.1371/journal.pone.0193210.].

摘要

[本文更正了文章的数字对象标识符：10.1371/journal.pone.0193210。]