相似文献
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GMF as an Actin Network Remodeling Factor.作为肌动蛋白网络重塑因子的 GMF。
Trends Cell Biol. 2018 Sep;28(9):749-760. doi: 10.1016/j.tcb.2018.04.008. Epub 2018 May 18.
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GMF is a cofilin homolog that binds Arp2/3 complex to stimulate filament debranching and inhibit actin nucleation.GMF 是一种与胞衬蛋白同源的蛋白，它可以结合 Arp2/3 复合物，从而刺激丝的分支解聚并抑制肌动蛋白成核。
Curr Biol. 2010 May 11;20(9):861-7. doi: 10.1016/j.cub.2010.03.026. Epub 2010 Apr 1.
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Glia maturation factor (GMF) interacts with Arp2/3 complex in a nucleotide state-dependent manner.神经胶质细胞成熟因子（GMF）以核苷酸状态依赖的方式与 Arp2/3 复合物相互作用。
J Biol Chem. 2013 Sep 6;288(36):25683-25688. doi: 10.1074/jbc.C113.493338. Epub 2013 Jul 29.
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Single-molecule analysis of actin filament debranching by cofilin and GMF.肌动蛋白丝分支解聚的单分子分析由束丝蛋白和 GMF 完成。
Proc Natl Acad Sci U S A. 2022 Jul 19;119(29):e2115129119. doi: 10.1073/pnas.2115129119. Epub 2022 Jul 11.
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GMF severs actin-Arp2/3 complex branch junctions by a cofilin-like mechanism.GMF 通过肌动蛋白样机制切断肌动蛋白-Arp2/3 复合物分支连接点。
Curr Biol. 2013 Jun 17;23(12):1037-45. doi: 10.1016/j.cub.2013.04.058. Epub 2013 May 30.
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Structural basis for regulation of Arp2/3 complex by GMF.GMF 调控 Arp2/3 复合物的结构基础。
Nat Struct Mol Biol. 2013 Sep;20(9):1062-8. doi: 10.1038/nsmb.2628. Epub 2013 Jul 28.
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Direct observation of cortactin protecting Arp2/3-actin filament branch junctions from GMF-mediated destabilization.直接观察到皮层肌动蛋白对Arp2/3-肌动蛋白丝分支连接点具有保护作用，使其免受GMF介导的去稳定作用。
Eur J Cell Biol. 2024 Mar;103(1):151378. doi: 10.1016/j.ejcb.2023.151378. Epub 2023 Dec 5.
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GMF is an evolutionarily developed Adf/cofilin-super family protein involved in the Arp2/3 complex-mediated organization of the actin cytoskeleton.GMF 是一个进化上发育的 Adf/cofilin-超家族蛋白，参与 Arp2/3 复合物介导的肌动蛋白细胞骨架的组织。
Cytoskeleton (Hoboken). 2010 Jun;67(6):373-82. doi: 10.1002/cm.20451.
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Abp1 promotes Arp2/3 complex-dependent actin nucleation and stabilizes branch junctions by antagonizing GMF.Abp1 通过拮抗 GMF 促进 Arp2/3 复合物依赖的肌动蛋白成核并稳定分支连接。
Nat Commun. 2018 Jul 24;9(1):2895. doi: 10.1038/s41467-018-05260-y.
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NMR solution structures of actin depolymerizing factor homology domains.肌动蛋白解聚因子同源结构域的 NMR 溶液结构。
Protein Sci. 2009 Nov;18(11):2384-92. doi: 10.1002/pro.248.
引用本文的文献
1
Two ligands of Arp2/3 complex, yeast coronin and GMF, interact and synergize in pruning branched actin networks.肌动蛋白相关蛋白2/3复合体（Arp2/3 complex）的两种配体，即酵母冠蛋白（yeast coronin）和GMF，在修剪分支状肌动蛋白网络中相互作用并协同发挥作用。
J Biol Chem. 2025 Mar;301(3):108191. doi: 10.1016/j.jbc.2025.108191. Epub 2025 Jan 16.
2
Interactions of VMAT2 with CDCrel-1 and Parkin in Methamphetamine Neurotoxicity.VMAT2与CDCrel-1和Parkin在甲基苯丙胺神经毒性中的相互作用。
Int J Mol Sci. 2024 Dec 5;25(23):13070. doi: 10.3390/ijms252313070.
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Glial maturation factor-β deficiency prevents oestrogen deficiency-induced bone loss by remodelling the actin network to suppress adipogenesis of bone marrow mesenchymal stem cells.胶质细胞成熟因子-β 缺乏通过重塑肌动蛋白网络抑制骨髓间充质干细胞成脂分化来预防雌激素缺乏诱导的骨丢失。
Cell Death Dis. 2024 Nov 14;15(11):829. doi: 10.1038/s41419-024-07234-z.
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Neurotoxic Methamphetamine Doses Alter CDCel-1 Levels and Its Interaction with Vesicular Monoamine Transporter-2 in Rat Striatum.神经毒性剂量的甲基苯丙胺会改变大鼠纹状体中CDCel-1的水平及其与囊泡单胺转运体-2的相互作用。
bioRxiv. 2024 Jul 23:2024.07.21.604458. doi: 10.1101/2024.07.21.604458.
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Bioinformatics-driven identification and validation of diagnostic biomarkers for cerebral ischemia reperfusion injury.生物信息学驱动的脑缺血再灌注损伤诊断生物标志物的鉴定与验证
Heliyon. 2024 Mar 31;10(7):e28565. doi: 10.1016/j.heliyon.2024.e28565. eCollection 2024 Apr 15.
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The stabilization of Arp2/3 complex generated actin filaments.Arp2/3 复合物稳定生成的肌动蛋白丝。
Biochem Soc Trans. 2024 Feb 28;52(1):343-352. doi: 10.1042/BST20230638.
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Comparative proteomics reveals the mechanism of cyclosporine production and mycelial growth in affected by different carbon sources.比较蛋白质组学揭示了不同碳源对环孢菌素产生和菌丝体生长的影响机制。
Front Microbiol. 2023 Dec 8;14:1259101. doi: 10.3389/fmicb.2023.1259101. eCollection 2023.
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Direct observation of cortactin protecting Arp2/3-actin filament branch junctions from GMF-mediated destabilization.直接观察到皮层肌动蛋白对Arp2/3-肌动蛋白丝分支连接点具有保护作用，使其免受GMF介导的去稳定作用。
Eur J Cell Biol. 2024 Mar;103(1):151378. doi: 10.1016/j.ejcb.2023.151378. Epub 2023 Dec 5.
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Mechanisms of actin disassembly and turnover.肌动蛋白解聚和周转率的机制。
J Cell Biol. 2023 Dec 4;222(12). doi: 10.1083/jcb.202309021. Epub 2023 Nov 10.
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High Level of GMFG Correlated to Poor Clinical Outcome and Promoted Cell Migration and Invasion through EMT Pathway in Triple-Negative Breast Cancer.GMFG 水平高与临床预后不良相关，并通过 EMT 通路促进三阴性乳腺癌细胞迁移和侵袭。
Genes (Basel). 2023 May 26;14(6):1157. doi: 10.3390/genes14061157.
本文引用的文献
1
Analysis of the interactions between GMF and Arp2/3 complex in two binding sites by molecular dynamics simulation.通过分子动力学模拟分析GMF与Arp2/3复合物在两个结合位点之间的相互作用。
Biochem Biophys Res Commun. 2018 Feb 5;496(2):529-535. doi: 10.1016/j.bbrc.2018.01.080. Epub 2018 Jan 12.
2
Phosphomimetic S3D cofilin binds but only weakly severs actin filaments.模拟磷酸化的S3D丝切蛋白能够结合肌动蛋白丝，但仅能微弱地切断它们。
J Biol Chem. 2017 Dec 1;292(48):19565-19579. doi: 10.1074/jbc.M117.808378. Epub 2017 Sep 22.
3
Structural Basis of Arp2/3 Complex Inhibition by GMF, Coronin, and Arpin.GMF、冠蛋白和Arpin对Arp2/3复合物的抑制作用的结构基础
J Mol Biol. 2017 Jan 20;429(2):237-248. doi: 10.1016/j.jmb.2016.11.030. Epub 2016 Dec 6.
4
Glia Maturation Factor-γ Regulates Monocyte Migration through Modulation of β1-Integrin.神经胶质成熟因子-γ通过调节β1整合素来调控单核细胞迁移。
J Biol Chem. 2016 Apr 15;291(16):8549-64. doi: 10.1074/jbc.M115.674200. Epub 2016 Feb 19.
5
Transgenic mice overexpressing glia maturation factor-β, an oxidative stress inducible gene, show premature aging due to Zmpste24 down-regulation.过表达胶质细胞成熟因子-β（一种氧化应激诱导基因）的转基因小鼠，由于Zmpste24下调而出现早衰。
Aging (Albany NY). 2015 Jul;7(7):486-99. doi: 10.18632/aging.100779.
6
Combinatorial genetic analysis of a network of actin disassembly-promoting factors.肌动蛋白解聚促进因子网络的组合遗传分析
Cytoskeleton (Hoboken). 2015 Jul;72(7):349-61. doi: 10.1002/cm.21231. Epub 2015 Aug 22.
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GMFβ controls branched actin content and lamellipodial retraction in fibroblasts.GMFβ控制成纤维细胞中分支肌动蛋白的含量和片状伪足的回缩。
J Cell Biol. 2015 Jun 22;209(6):803-12. doi: 10.1083/jcb.201501094.
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Single-molecule imaging of a three-component ordered actin disassembly mechanism.一种三组分有序肌动蛋白解聚机制的单分子成像
Nat Commun. 2015 May 21;6:7202. doi: 10.1038/ncomms8202.
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Actin flows mediate a universal coupling between cell speed and cell persistence.肌动蛋白流介导了细胞速度和细胞持续时间之间的普遍耦合。
Cell. 2015 Apr 9;161(2):374-86. doi: 10.1016/j.cell.2015.01.056. Epub 2015 Mar 19.
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A novel role for WAVE1 in controlling actin network growth rate and architecture.WAVE1在控制肌动蛋白网络生长速率和结构方面的新作用。
Mol Biol Cell. 2015 Feb 1;26(3):495-505. doi: 10.1091/mbc.E14-10-1477. Epub 2014 Dec 3.
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