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Author Correction: HIV-1 counteracts an innate restriction by amyloid precursor protein resulting in neurodegeneration.作者更正:HIV-1通过淀粉样前体蛋白对抗一种先天性限制,导致神经退行性变。
Nat Commun. 2018 Jun 5;9(1):2257. doi: 10.1038/s41467-018-04750-3.
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Prescription of Controlled Substances: Benefits and Risks管制药品的处方:益处与风险
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引用本文的文献

1
Inflammatory responses revealed through HIV infection of microglia-containing cerebral organoids.通过含小胶质细胞的脑类器官感染HIV揭示的炎症反应。
J Neuroinflammation. 2025 Feb 10;22(1):36. doi: 10.1186/s12974-025-03353-2.
2
HIV-1 Tat-mediated astrocytic amyloidosis involves the HIF-1α/lncRNA BACE1-AS axis.HIV-1 Tat 介导的星形胶质细胞淀粉样变性涉及 HIF-1α/lncRNA BACE1-AS 轴。
PLoS Biol. 2020 May 26;18(5):e3000660. doi: 10.1371/journal.pbio.3000660. eCollection 2020 May.

作者更正:HIV-1通过淀粉样前体蛋白对抗一种先天性限制,导致神经退行性变。

Author Correction: HIV-1 counteracts an innate restriction by amyloid precursor protein resulting in neurodegeneration.

作者信息

Chai Qingqing, Jovasevic Vladimir, Malikov Viacheslav, Sabo Yosef, Morham Scott, Walsh Derek, Naghavi Mojgan H

机构信息

Department of Microbiology-Immunology, Northwestern University Feinberg School of Medicine, Chicago, IL, 60611, USA.

Department of Biochemistry and Molecular Biophysics, Howard Hughes Medical Institute, Columbia University, New York, NY, 10032, USA.

出版信息

Nat Commun. 2018 Jun 5;9(1):2257. doi: 10.1038/s41467-018-04750-3.

DOI:10.1038/s41467-018-04750-3
PMID:29872052
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC5988785/
Abstract

The original version of this Article contained an error in the Methods section 'Viruses and drugs'. The timing for drug treatment of CHME3 4 × 4 or 293T cells with γ-secretase inhibitor or BACE1 inhibitor was incorrectly given as '1 day prior to infection or transfection' and should have stated '4 or 6 h post transfection or infection, respectively'. This error is now corrected in both the PDF and HTML versions of the Article.

摘要

本文的原始版本在“病毒与药物”方法部分存在一处错误。用γ-分泌酶抑制剂或β-分泌酶1抑制剂对CHME3 4×4或293T细胞进行药物处理的时间错误地写成了“感染或转染前1天”,而应该分别表述为“转染或感染后4或6小时”。此错误现已在本文的PDF和HTML版本中更正。