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Ag-specific CD4 T cells promote innate immune responses in liver ischemia reperfusion injury.

作者信息

Rao Jianhua, Cheng Feng, Yang Shikun, Zhai Yuan, Lu Ling

机构信息

Hepatobiliary/Liver Transplantation Center, The First Affiliated Hospital of Nanjing Medical University, Nanjing, 210029, China.

Dumont-UCLA Transplant Center, Division of Liver and Pancreas Transplantation, Department of Surgery, David Geffen School of Medicine at University of California-Los Angeles, Los Angeles, CA, USA.

出版信息

Cell Mol Immunol. 2019 Jan;16(1):98-100. doi: 10.1038/s41423-018-0051-x. Epub 2018 Jun 15.

DOI:10.1038/s41423-018-0051-x
PMID:29907880
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC6318264/
Abstract
摘要

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Cell Mol Immunol. 2019 Jan;16(1):98-100. doi: 10.1038/s41423-018-0051-x. Epub 2018 Jun 15.
2
CXCR3+CD4+ T cells mediate innate immune function in the pathophysiology of liver ischemia/reperfusion injury.CXCR3 + CD4 + T细胞在肝脏缺血/再灌注损伤的病理生理学中介导先天性免疫功能。
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Am J Physiol Gastrointest Liver Physiol. 2005 Nov;289(5):G969-76. doi: 10.1152/ajpgi.00223.2005. Epub 2005 Jul 7.
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Modulating CD4+ T cell migration in the postischemic liver: hepatic stellate cells as new therapeutic target?调节缺血后肝脏中CD4+ T细胞的迁移：肝星状细胞作为新的治疗靶点？
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CD154-CD40 T-cell costimulation pathway is required in the mechanism of hepatic ischemia/reperfusion injury, and its blockade facilitates and depends on heme oxygenase-1 mediated cytoprotection.CD154-CD40 T细胞共刺激途径在肝缺血/再灌注损伤机制中是必需的，其阻断促进并依赖于血红素加氧酶-1介导的细胞保护作用。
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A novel interpretation of immune redundancy and duality in reperfusion injury with important implications for intervention in ischaemic disease.对再灌注损伤中免疫冗余和二元性的一种新解释，对缺血性疾病的干预具有重要意义。
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Contribution of CD4+ T cells to the early mechanisms of ischemia- reperfusion injury in a mouse model of acute renal failure.CD4 + T细胞在急性肾衰竭小鼠模型缺血再灌注损伤早期机制中的作用。
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Eur J Med Res. 2024 Jun 13;29(1):326. doi: 10.1186/s40001-024-01928-y.
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FOXO1 regulates Th17 cell-mediated hepatocellular carcinoma recurrence after hepatic ischemia-reperfusion injury.FOXO1 调控肝缺血再灌注损伤后 Th17 细胞介导的肝细胞癌复发。
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Adv Sci (Weinh). 2020 Aug 20;7(18):1903746. doi: 10.1002/advs.201903746. eCollection 2020 Sep.
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Acidic Microenvironment Regulates the Severity of Hepatic Ischemia/Reperfusion Injury by Modulating the Generation and Function of Tregs via the PI3K-mTOR Pathway.酸性微环境通过 PI3K-mTOR 通路调节 Tregs 的生成和功能来调节肝缺血/再灌注损伤的严重程度。
Front Immunol. 2020 Jan 9;10:2945. doi: 10.3389/fimmu.2019.02945. eCollection 2019.

本文引用的文献

1
Contribution of Toll-like receptors to the control of hepatitis B virus infection by initiating antiviral innate responses and promoting specific adaptive immune responses.Toll样受体通过启动抗病毒固有免疫反应和促进特异性适应性免疫反应对控制乙型肝炎病毒感染的作用。
Cell Mol Immunol. 2015 May;12(3):273-82. doi: 10.1038/cmi.2014.112. Epub 2014 Nov 24.
2
Regulatory T cells as immunotherapy.调节性T细胞作为免疫疗法。
Front Immunol. 2014 Feb 11;5:46. doi: 10.3389/fimmu.2014.00046. eCollection 2014.
3
Altered inactivation of commensal LPS due to acyloxyacyl hydrolase deficiency in colonic dendritic cells impairs mucosal Th17 immunity.由于结肠树突状细胞中酰氧基酰基水解酶缺乏导致共生 LPS 失活改变，从而损害黏膜 Th17 免疫。
Proc Natl Acad Sci U S A. 2014 Jan 7;111(1):373-8. doi: 10.1073/pnas.1311987111. Epub 2013 Dec 16.
4
Control of in vivo collateral damage generated by T cell immunity.控制 T 细胞免疫引起的体内侧支损伤。
J Immunol. 2013 Aug 15;191(4):1686-91. doi: 10.4049/jimmunol.1203240. Epub 2013 Jul 12.
5
Autocrine adenosine signaling promotes regulatory T cell-mediated renal protection.自分泌腺苷信号促进调节性 T 细胞介导的肾脏保护。
J Am Soc Nephrol. 2012 Sep;23(9):1528-37. doi: 10.1681/ASN.2012010070. Epub 2012 Jul 26.
6
Apoptotic cells attenuate fulminant hepatitis by priming Kupffer cells to produce interleukin-10 through membrane-bound TGF-β.凋亡细胞通过膜结合 TGF-β使枯否细胞产生白细胞介素-10从而减轻暴发性肝炎。
Hepatology. 2011 Jan;53(1):306-16. doi: 10.1002/hep.24029. Epub 2010 Dec 7.
7
Alloimmune activation enhances innate tissue inflammation/injury in a mouse model of liver ischemia/reperfusion injury.同种免疫激活增强了小鼠肝缺血/再灌注损伤模型中的固有组织炎症/损伤。
Am J Transplant. 2010 Aug;10(8):1729-37. doi: 10.1111/j.1600-6143.2010.03205.x.
8
CD4 T cells promote tissue inflammation via CD40 signaling without de novo activation in a murine model of liver ischemia/reperfusion injury.在小鼠肝脏缺血/再灌注损伤模型中，CD4 T细胞通过CD40信号传导促进组织炎症，而无需从头激活。
Hepatology. 2009 Nov;50(5):1537-46. doi: 10.1002/hep.23153.
9
Type I, but not type II, interferon is critical in liver injury induced after ischemia and reperfusion.I型干扰素而非II型干扰素在缺血再灌注后诱导的肝损伤中起关键作用。
Hepatology. 2008 Jan;47(1):199-206. doi: 10.1002/hep.21970.
10
Kupffer cell-derived interleukin 10 is responsible for impaired bacterial clearance in bile duct-ligated mice.库普弗细胞衍生的白细胞介素10导致胆管结扎小鼠的细菌清除受损。
Hepatology. 2004 Aug;40(2):414-23. doi: 10.1002/hep.20301.