Hambræus Johan, Westergren Hans
Smärtkliniken i Umeå, Kungsgatan 42, 903 25 Umeå, Sweden.
Section for Specialized Pain Rehabilitation, Department of Rehabilitation Medicine, Skåne University Hospital and Department of Health Sciences, Lund University, Skåne, Sweden.
Scand J Pain. 2012 Oct 1;3(4):228-240. doi: 10.1016/j.sjpain.2012.07.002.
Introduction Patients exposed to whiplash trauma are at risk of developing pain and dysfunction of the neck and shoulder. Although rarely discussed in the literature, some patients also develop autonomic dysfunction. Case presentation A previously healthy 41-year-old woman was involved in a "head-on" car crash. During the following 3 years she developed severe and complex post-traumatic pain syndrome, which consisted of neck pain, lumbar pain, sensory-motor dysfunction, and myoclonic muscular contractions. Despite pharmacotherapy, physiotherapy, and rehabilitation, her condition worsened, resulting in severe disability. Fourteen years after the car crash, an interventional pain therapy program was started, which consisted of sympathetic ganglion impar block and medial branch blocks of facet joints at different levels. These treatment strategies ultimately normalized her sensory-motor dysfunction, reduced her autonomic dysfunction, and stopped the myoclonic muscular contractions. Conclusion This case highlights a possible interaction between the pain-generating facet joints, the somatosensory nervous system, and the autonomic/sympathetic nervous systems. The case also highlights the importance of identifying autonomic dysfunction in patients with persisting pain syndromes. Implications This complex case shows that many clinical phenomena cannot be explained using our present knowledge of pain mechanisms. We hope that readers who have observed similar cases can learn from our case, and are encouraged to publish their observations.
遭受挥鞭样创伤的患者有发生颈部和肩部疼痛及功能障碍的风险。尽管文献中很少讨论,但一些患者还会出现自主神经功能障碍。
一名41岁的既往健康女性遭遇了“正面”车祸。在接下来的3年里,她患上了严重且复杂的创伤后疼痛综合征,包括颈部疼痛、腰部疼痛、感觉运动功能障碍和肌阵挛性肌肉收缩。尽管接受了药物治疗、物理治疗和康复治疗,她的病情仍恶化,导致严重残疾。车祸发生14年后,启动了一项介入性疼痛治疗方案,包括奇神经节阻滞和不同节段小关节内侧支阻滞。这些治疗策略最终使她的感觉运动功能障碍恢复正常,减轻了自主神经功能障碍,并停止了肌阵挛性肌肉收缩。
该病例突出了产生疼痛的小关节、躯体感觉神经系统和自主神经/交感神经系统之间可能存在的相互作用。该病例还强调了在持续性疼痛综合征患者中识别自主神经功能障碍的重要性。
这个复杂的病例表明,许多临床现象无法用我们目前对疼痛机制的认识来解释。我们希望观察到类似病例的读者能从我们的病例中吸取经验,并受到鼓励发表他们的观察结果。