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Herpes simplex virus latency: adaptation to the peripheral nervous system. II.

作者信息

Price R W

出版信息

Cancer Invest. 1985;3(4):389-403. doi: 10.3109/07357908509039799.

DOI:10.3109/07357908509039799
PMID:2992726
Abstract
摘要

相似文献

1
Herpes simplex virus latency: adaptation to the peripheral nervous system. II.
Cancer Invest. 1985;3(4):389-403. doi: 10.3109/07357908509039799.
2
Herpes simplex virus latency: adaptation to the peripheral nervous system. I.
Cancer Invest. 1985;3(3):285-92. doi: 10.3109/07357908509039789.
3
Transcripts associated with herpes simplex virus latency.与单纯疱疹病毒潜伏相关的转录本。
Adv Exp Med Biol. 1990;278:199-204. doi: 10.1007/978-1-4684-5853-4_20.
4
[Latency of herpes simplex virus. Current knowledge].[单纯疱疹病毒的潜伏期。当前认知]
Enferm Infecc Microbiol Clin. 1991 Mar;9(3):172-6.
5
Herpes simplex virus latency.
Clin Dermatol. 1984 Apr-Jun;2(2):46-55. doi: 10.1016/0738-081x(84)90065-8.
6
The continuing problem of herpes simplex virus persistence.单纯疱疹病毒持续存在的持续性问题。
Acta Virol. 1983 Sep;27(5):442-50.
7
Viral, neuronal and immune factors which may influence herpes simplex virus (HSV) latency and reactivation.可能影响单纯疱疹病毒(HSV)潜伏和再激活的病毒、神经元及免疫因素。
Microb Pathog. 1993 Aug;15(2):83-91. doi: 10.1006/mpat.1993.1059.
8
The weakly virulent herpes simplex virus type 1 strain KOS-63 establishes peripheral and central nervous system latency following intranasal infection of rabbits, but poorly reactivates in vivo.低毒力的1型单纯疱疹病毒株KOS-63经鼻内感染兔后可在周围和中枢神经系统建立潜伏感染,但在体内再激活能力较弱。
J Neuropathol Exp Neurol. 1992 Sep;51(5):550-9. doi: 10.1097/00005072-199209000-00010.
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Repression of viral transcription during herpes simplex virus latency.单纯疱疹病毒潜伏期间病毒转录的抑制
J Gen Virol. 2000 Jan;81(Pt 1):1-19. doi: 10.1099/0022-1317-81-1-1.
10
[Mechanisms involved in latent herpes simplex virus infection. I. In vivo studies].[潜伏性单纯疱疹病毒感染的相关机制。I. 体内研究]
Uirusu. 1985 Jun;35(1):1-10. doi: 10.2222/jsv.35.1.

引用本文的文献

1
Thymidine kinase-negative herpes simplex virus mutants establish latency in mouse trigeminal ganglia but do not reactivate.胸苷激酶阴性单纯疱疹病毒突变体可在小鼠三叉神经节中建立潜伏感染,但不会再激活。
Proc Natl Acad Sci U S A. 1989 Jun;86(12):4736-40. doi: 10.1073/pnas.86.12.4736.
2
A conserved open reading frame that overlaps the herpes simplex virus thymidine kinase gene is important for viral growth in cell culture.一个与单纯疱疹病毒胸苷激酶基因重叠的保守开放阅读框对病毒在细胞培养中的生长很重要。
J Virol. 1989 Apr;63(4):1839-43. doi: 10.1128/JVI.63.4.1839-1843.1989.
3
Regulation and cell-type-specific activity of a promoter located upstream of the latency-associated transcript of herpes simplex virus type 1.
1型单纯疱疹病毒潜伏相关转录本上游启动子的调控及细胞类型特异性活性
J Virol. 1990 Jul;64(7):3269-79. doi: 10.1128/JVI.64.7.3269-3279.1990.
4
Localization of cis-acting sequence requirements in the promoter of the latency-associated transcript of herpes simplex virus type 1 required for cell-type-specific activity.
J Virol. 1992 Jun;66(6):3573-82. doi: 10.1128/JVI.66.6.3573-3582.1992.