可能影响单纯疱疹病毒（HSV）潜伏和再激活的病毒、神经元及免疫因素。 - Suppr

Suppr

超能文献

Viral, neuronal and immune factors which may influence herpes simplex virus (HSV) latency and reactivation.

作者信息

Fraser N W, Valyi-Nagy T

机构信息

Wistar Institute, Philadelphia, PA 19104.

出版信息

Microb Pathog. 1993 Aug;15(2):83-91. doi: 10.1006/mpat.1993.1059.

DOI:10.1006/mpat.1993.1059

PMID:8255209

Abstract

摘要

相似文献

Viral, neuronal and immune factors which may influence herpes simplex virus (HSV) latency and reactivation.可能影响单纯疱疹病毒（HSV）潜伏和再激活的病毒、神经元及免疫因素。

Microb Pathog. 1993 Aug;15(2):83-91. doi: 10.1006/mpat.1993.1059.

[Molecular biology of pathogenesis by herpesvirus].[疱疹病毒致病机制的分子生物学]

Rev Latinoam Microbiol. 1991 Jan-Mar;33(1):77-85.

Neuronal control of herpes simplex virus latency.单纯疱疹病毒潜伏的神经元控制

Virology. 1993 Aug;195(2):337-47. doi: 10.1006/viro.1993.1384.

[Latency of herpes simplex virus. Current knowledge].[单纯疱疹病毒的潜伏期。当前认知]

Enferm Infecc Microbiol Clin. 1991 Mar;9(3):172-6.

The continuing problem of herpes simplex virus persistence.单纯疱疹病毒持续存在的持续性问题。

Acta Virol. 1983 Sep;27(5):442-50.

[Herpes simplex virus: mechanism of latency and reactivation phases].[单纯疱疹病毒：潜伏和再激活阶段的机制]

Ann Dermatol Venereol. 1997;124(5):409-15.

Chromatin control of herpes simplex virus lytic and latent infection.单纯疱疹病毒裂解性感染和潜伏感染的染色质调控

Nat Rev Microbiol. 2008 Mar;6(3):211-21. doi: 10.1038/nrmicro1794.

Immunity to latent viral infection: many skirmishes but few fatalities.对潜伏性病毒感染的免疫：多次小规模冲突但鲜有致命情况。

Trends Immunol. 2004 May;25(5):230-4. doi: 10.1016/j.it.2004.02.010.

[Topics on herpes simplex virus pathogenesis--roles of viral 'non-essential' genes and host immune status].[单纯疱疹病毒发病机制的相关主题——病毒“非必需”基因和宿主免疫状态的作用]

Uirusu. 2002 Jun;52(1):109-15.

[Present status and task the in study of pathophysiologic mechanism of latent herpes simplex virus infection and the viral re-activation].[单纯疱疹病毒潜伏感染及病毒再激活病理生理机制的研究现状与任务]

Fukuoka Igaku Zasshi. 1995 Jul;86(7):299-303.

引用本文的文献

In the Absence of Type-1 IFN, HSV-1 LAT Increases γ34.5 Expression and Enhances Mortality in Infected Mice.在缺乏I型干扰素的情况下，单纯疱疹病毒1型潜伏相关转录物增加γ34.5的表达并提高感染小鼠的死亡率。

Viruses. 2025 Jul 29;17(8):1061. doi: 10.3390/v17081061.

IFNβ absence compensates for LAT functions in latency reactivation and T cell exhaustion.IFNβ缺失可补偿LAT在潜伏激活和T细胞耗竭中的功能。

J Virol. 2025 Jun 17;99(6):e0037425. doi: 10.1128/jvi.00374-25. Epub 2025 May 12.

The anti-apoptotic function of HSV-1 LAT in neuronal cell cultures but not its function during reactivation correlates with expression of two small non-coding RNAs, sncRNA1&2.单纯疱疹病毒 1 潜伏相关转录物在神经元细胞培养中的抗细胞凋亡功能与其在再激活过程中的功能无关，与其表达的两种小非编码 RNA（sncRNA1 和 sncRNA2）相关。

PLoS Pathog. 2024 Jun 10;20(6):e1012307. doi: 10.1371/journal.ppat.1012307. eCollection 2024 Jun.

Binding of herpesvirus entry mediator (HVEM) and HSV-1 gD affect reactivation but not latency levels.疱疹病毒进入介质（HVEM）与单纯疱疹病毒1型糖蛋白D（HSV-1 gD）的结合影响病毒再激活，但不影响潜伏水平。

PLoS Pathog. 2023 Sep 22;19(9):e1011693. doi: 10.1371/journal.ppat.1011693. eCollection 2023 Sep.

Stacking the odds: Multiple sites for HSV-1 latency.病毒潜伏的多重据点：疱疹病毒 1 型。

Sci Adv. 2023 Jan 25;9(4):eadf4904. doi: 10.1126/sciadv.adf4904.

Knockout of signal peptide peptidase in the eye reduces HSV-1 replication and eye disease in ocularly infected mice.敲除眼部信号肽肽酶可减少眼感染小鼠中 HSV-1 的复制和眼部疾病。

PLoS Pathog. 2022 Oct 10;18(10):e1010898. doi: 10.1371/journal.ppat.1010898. eCollection 2022 Oct.

Host Molecules That Promote Pathophysiology of Ocular Herpes.促进眼部疱疹病理生理学的宿主分子。

Front Microbiol. 2022 Jan 25;13:818658. doi: 10.3389/fmicb.2022.818658. eCollection 2022.

Absence of signal peptide peptidase in peripheral sensory neurons affects latency-reactivation in HSV-1 ocularly infected mice.外周感觉神经元中信号肽肽酶的缺失影响单纯疱疹病毒1型眼部感染小鼠的潜伏再激活。

PLoS Pathog. 2022 Jan 31;18(1):e1010281. doi: 10.1371/journal.ppat.1010281. eCollection 2022 Jan.

Absence of CD28-CTLA4-PD-L1 Costimulatory Molecules Reduces Herpes Simplex Virus 1 Reactivation.CD28-CTLA4-PD-L1 共刺激分子缺失可减少单纯疱疹病毒 1 复发。

mBio. 2021 Aug 31;12(4):e0117621. doi: 10.1128/mBio.01176-21. Epub 2021 Jul 20.

Role of Herpes Simplex Virus Type 1 (HSV-1) Glycoprotein K (gK) Pathogenic CD8 T Cells in Exacerbation of Eye Disease.单纯疱疹病毒 1 型（HSV-1）糖蛋白 K（gK）致病性 CD8 T 细胞在眼部疾病恶化中的作用。

Front Immunol. 2018 Dec 7;9:2895. doi: 10.3389/fimmu.2018.02895. eCollection 2018.

文献AI研究员

20分钟写一篇综述，助力文献阅读效率提升50倍。

立即体验

用中文搜PubMed

大模型驱动的PubMed中文搜索引擎

马上搜索

文档翻译

学术文献翻译模型，支持多种主流文档格式。

立即体验