Allure Laser Center & Medispa, Kirkland.
Department of Ophthalmology, University of Washington.
J Glaucoma. 2018 Oct;27(10):e154-e157. doi: 10.1097/IJG.0000000000001021.
Trans-lamina cribrosa pressure has been postulated to be a contributor in the development of a glaucomatous optic nerve versus optic nerve edema, depending on the pressure gradient. Uncertainty remains in the therapeutic outcome of adjusting this gradient.
We discuss a unique case of idiopathic intracranial hypertension presenting as asymmetric optic disc edema following trabeculectomy. It was treated via optic nerve fenestration due to the patient remaining symptomatic on maximum tolerated acetazolamide. Intraocular pressure stabilized into target range and the optic nerve edema resolved.
Rarely, intraocular pressure reduction can unmask elevated intracranial pressure, leading to optic nerve edema. Optic nerve sheath fenestration is a practical therapeutic modality to consider when treating this occurrence.
根据压力梯度的不同，跨筛板压力被认为是青光眼视神经病变与视神经水肿发展的一个影响因素。在调整这种梯度的治疗效果方面仍存在不确定性。
我们讨论了一例特发性颅内高压的独特病例，该患者在小梁切除术 后出现不对称性视盘水肿。由于患者在最大耐受乙酰唑胺治疗后仍有症状，因此通过视神经开窗术进行治疗。眼内压稳定在目标范围内，视神经水肿消退。
很少有情况下，眼压降低会导致颅内压升高，从而导致视神经水肿。当治疗这种情况时，视神经鞘开窗术是一种实用的治疗方法。
