对《Nup42与IP协同调控Gle1对Dbp5/DDX19B的刺激以促进酵母和人类细胞中的mRNA输出》的更正 - Suppr | 超能文献

Correction to: Nup42 and IP coordinate Gle1 stimulation of Dbp5/DDX19B for mRNA export in yeast and human cells.

作者信息

Adams Rebecca L, Mason Aaron C, Glass Laura, Wente Susan R

机构信息

Department of Cell and Developmental Biology, Vanderbilt University School of Medicine, Nashville, Tennessee.

出版信息

Traffic. 2018 Aug;19(8):650. doi: 10.1111/tra.12585.

DOI:10.1111/tra.12585

Abstract

摘要

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Correction to: Nup42 and IP coordinate Gle1 stimulation of Dbp5/DDX19B for mRNA export in yeast and human cells.对《Nup42与IP协同调控Gle1对Dbp5/DDX19B的刺激以促进酵母和人类细胞中的mRNA输出》的更正

Traffic. 2018 Aug;19(8):650. doi: 10.1111/tra.12585.

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Nup42 and IP coordinate Gle1 stimulation of Dbp5/DDX19B for mRNA export in yeast and human cells.Nup42 和 IP 共同协调 Gle1 对酵母和人细胞中 Dbp5/DDX19B mRNA 输出的刺激作用。

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Depletion of mRNA export regulator DBP5/DDX19, GLE1 or IPPK that is a key enzyme for the production of IP6, resulting in differentially altered cytoplasmic mRNA expression and specific cell defect.mRNA 输出调节因子 DBP5/DDX19、GLE1 或 IPPK 的耗竭，这些因子是 IP6 产生的关键酶，导致细胞质 mRNA 表达的差异改变和特定的细胞缺陷。

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Activation of the DExD/H-box protein Dbp5 by the nuclear-pore protein Gle1 and its coactivator InsP6 is required for mRNA export.核孔蛋白Gle1及其共激活因子InsP6对DExD/H盒蛋白Dbp5的激活是mRNA输出所必需的。

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Gle1 is required for tRNA to stimulate Dbp5 ATPase activity and to promote Dbp5 mediated tRNA export .Gle1是tRNA刺激Dbp5 ATP酶活性并促进Dbp5介导的tRNA输出所必需的。

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Correction: Depletion of mRNA export regulator DBP5/DDX19, GLE1 or IPPK that is a key enzyme for the production of IP6, resulting in differentially altered cytoplasmic mRNA expression and specific cell defect.修正：mRNA 输出调节因子 DBP5/DDX19、GLE1 或 IPPK（一种产生肌醇六磷酸的关键酶）的缺失，导致细胞质 mRNA 表达的差异改变和特定细胞缺陷。

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Dbp5 - from nuclear export to translation.Dbp5——从核输出到翻译

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InsP6-sensitive variants of the Gle1 mRNA export factor rescue growth and fertility defects of the ipk1 low-phytic-acid mutation in Arabidopsis.Gle1 mRNA输出因子的肌醇六磷酸敏感变体挽救了拟南芥中ipk1低植酸突变的生长和育性缺陷。

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