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Correction to: Nup42 and IP coordinate Gle1 stimulation of Dbp5/DDX19B for mRNA export in yeast and human cells.

作者信息

Adams Rebecca L, Mason Aaron C, Glass Laura, Wente Susan R

机构信息

Department of Cell and Developmental Biology, Vanderbilt University School of Medicine, Nashville, Tennessee.

出版信息

Traffic. 2018 Aug;19(8):650. doi: 10.1111/tra.12585.

DOI:10.1111/tra.12585
PMID:29999599
Abstract
摘要

相似文献

1
Correction to: Nup42 and IP coordinate Gle1 stimulation of Dbp5/DDX19B for mRNA export in yeast and human cells.对《Nup42与IP协同调控Gle1对Dbp5/DDX19B的刺激以促进酵母和人类细胞中的mRNA输出》的更正
Traffic. 2018 Aug;19(8):650. doi: 10.1111/tra.12585.
2
Nup42 and IP coordinate Gle1 stimulation of Dbp5/DDX19B for mRNA export in yeast and human cells.Nup42 和 IP 共同协调 Gle1 对酵母和人细胞中 Dbp5/DDX19B mRNA 输出的刺激作用。
Traffic. 2017 Dec;18(12):776-790. doi: 10.1111/tra.12526. Epub 2017 Oct 16.
3
Depletion of mRNA export regulator DBP5/DDX19, GLE1 or IPPK that is a key enzyme for the production of IP6, resulting in differentially altered cytoplasmic mRNA expression and specific cell defect.mRNA 输出调节因子 DBP5/DDX19、GLE1 或 IPPK 的耗竭,这些因子是 IP6 产生的关键酶,导致细胞质 mRNA 表达的差异改变和特定的细胞缺陷。
PLoS One. 2018 May 10;13(5):e0197165. doi: 10.1371/journal.pone.0197165. eCollection 2018.
4
Inositol hexakisphosphate and Gle1 activate the DEAD-box protein Dbp5 for nuclear mRNA export.肌醇六磷酸和Gle1激活DEAD盒蛋白Dbp5以促进核mRNA输出。
Nat Cell Biol. 2006 Jul;8(7):711-6. doi: 10.1038/ncb1427. Epub 2006 Jun 18.
5
Activation of the DExD/H-box protein Dbp5 by the nuclear-pore protein Gle1 and its coactivator InsP6 is required for mRNA export.核孔蛋白Gle1及其共激活因子InsP6对DExD/H盒蛋白Dbp5的激活是mRNA输出所必需的。
Nat Cell Biol. 2006 Jul;8(7):668-76. doi: 10.1038/ncb1424. Epub 2006 Jun 18.
6
Gle1 is required for tRNA to stimulate Dbp5 ATPase activity and to promote Dbp5 mediated tRNA export .Gle1是tRNA刺激Dbp5 ATP酶活性并促进Dbp5介导的tRNA输出所必需的。
bioRxiv. 2023 Nov 9:2023.06.29.547072. doi: 10.1101/2023.06.29.547072.
7
Correction: Depletion of mRNA export regulator DBP5/DDX19, GLE1 or IPPK that is a key enzyme for the production of IP6, resulting in differentially altered cytoplasmic mRNA expression and specific cell defect.修正:mRNA 输出调节因子 DBP5/DDX19、GLE1 或 IPPK(一种产生肌醇六磷酸的关键酶)的缺失,导致细胞质 mRNA 表达的差异改变和特定细胞缺陷。
PLoS One. 2019 Jul 25;14(7):e0220511. doi: 10.1371/journal.pone.0220511. eCollection 2019.
8
Dbp5 - from nuclear export to translation.Dbp5——从核输出到翻译
Biochim Biophys Acta. 2013 Aug;1829(8):791-8. doi: 10.1016/j.bbagrm.2012.10.010. Epub 2012 Nov 2.
9
Nuclear Export of Pre-Ribosomal Subunits Requires Dbp5, but Not as an RNA-Helicase as for mRNA Export.核糖体前体亚基的核输出需要Dbp5,但不像mRNA输出那样作为一种RNA解旋酶。
PLoS One. 2016 Feb 12;11(2):e0149571. doi: 10.1371/journal.pone.0149571. eCollection 2016.
10
InsP6-sensitive variants of the Gle1 mRNA export factor rescue growth and fertility defects of the ipk1 low-phytic-acid mutation in Arabidopsis.Gle1 mRNA输出因子的肌醇六磷酸敏感变体挽救了拟南芥中ipk1低植酸突变的生长和育性缺陷。
Plant Cell. 2015 Feb;27(2):417-31. doi: 10.1105/tpc.114.132134. Epub 2015 Feb 10.

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