Hauge Tobias, Kleven Ole Christian, Johnson Egil, Hofstad Bjørn, Johannessen Hans-Olaf
a Department of Surgery , Drammen Hospital, Vestre Viken HF , Drammen , Norway.
b Department of Surgery , Lillehammer Hospital, Sykehuset Innlandet , Lillehammer , Norway.
Scand J Gastroenterol. 2018 Aug;53(8):905-909. doi: 10.1080/00365521.2018.1495760.
Food bolus-induced esophageal perforation is much more seldom than iatrogenic and emetic esophageal rupture. We present results from a non-operative treatment approach as well as long-term functional outcome.
Medical records of 10 consecutive patients with food bolus-induced esophageal perforation from October 2007 to May 2015 were retrospectively registered in a database. Six patients developed perforation related to endoscopic removal of impacted food, and four during esophageal passage of bone, meat or bread. Treatment was sealing the perforation by stenting (n = 7) with (n = 4) or without (n = 3) chest tube drainage, chest tube drainage (n = 1), observation (n = 1) and gastroesophageal resection (n = 1) because of concomitant emesis of gastric effluent. After median 51 months nine patients reported about dysphagia, fatigue and health-related quality of life.
Ten patients aged median 62.5 years (range 30-85) stayed in our hospital for 12 days (5-68 days). There was no treatment-related mortality. Nine patients were alive 63 months (18-126) after perforation. Five needed restenting (leakage, migration, impacted stent), two had drainage of a mediastinal abscess, one patient developed a transient esophagobronchial fistula. Dysphagia score was 0 (0-1). One patient developed dysphagia for some solid food. Scores for fatigue and HRQoL was similar to a Norwegian reference population.
Treatment mainly with a non-operative approach occurred without mortality. Complications were handled by restenting and abscess drainage. Functional result for dysphagia was excellent. Interesting results on fatigue and HRQoL must be interpreted with caution because of a limited patient material.
食物团块所致食管穿孔远比医源性和呕吐性食管破裂少见。我们展示了非手术治疗方法的结果以及长期功能结局。
回顾性收集2007年10月至2015年5月连续10例食物团块所致食管穿孔患者的病历资料并录入数据库。6例患者因内镜下取出嵌顿食物发生穿孔,4例在食管通过骨头、肉类或面包时发生穿孔。治疗方法包括置入支架封闭穿孔(7例),其中4例同时行胸腔闭式引流,3例未行胸腔闭式引流,1例行胸腔闭式引流,1例观察,1例因伴有胃内容物呕吐而行胃食管切除术。中位随访51个月后,9例患者报告了吞咽困难、疲劳及健康相关生活质量情况。
10例患者年龄中位数为62.5岁(范围30 - 85岁),在我院住院12天(5 - 68天)。无治疗相关死亡。9例患者穿孔后63个月(18 - 126个月)仍存活。5例需要再次置入支架(渗漏、移位、支架嵌顿),2例纵隔脓肿引流,1例患者出现短暂性食管支气管瘘。吞咽困难评分为0(0 - 1)。1例患者对某些固体食物出现吞咽困难。疲劳及健康相关生活质量评分与挪威参考人群相似。
主要采用非手术方法治疗,无死亡病例。并发症通过再次置入支架和脓肿引流处理。吞咽困难的功能结局良好。由于病例数量有限,疲劳及健康相关生活质量方面的有趣结果必须谨慎解读。