Witmer R
Klin Monbl Augenheilkd. 1977 Jun;170(6):837-42.
The secondary rise of i.o. pressure in uveitis may lead to a true secondary glaucoma or to hypertensive uveitis. The etiology of the endogenous inflammation does not seem to play a role. Pathogenetically the occlusion of the pupil with the formation of iris bombé and the obliteration of the chamber angle by exudate are important factors, while the hypersecretion of aqueous humor plays a minor role. Medical treatment consists in mydriatics and steroids. Surgical treatment depends on the pathogenetic mechanism and consists either in sector iridectomy or a filtering procedure.
葡萄膜炎中眼内压的继发性升高可能导致真正的继发性青光眼或高血压性葡萄膜炎。内源性炎症的病因似乎不起作用。从发病机制来看，瞳孔阻塞伴虹膜膨隆形成以及房角被渗出物闭塞是重要因素，而房水分泌过多起的作用较小。药物治疗包括使用散瞳剂和类固醇。手术治疗取决于发病机制，包括扇形虹膜切除术或滤过手术。
