Ritch R
Trans Ophthalmol Soc U K (1962). 1981 Sep;101 (Pt 3)(3):321-4.
Glaucoma secondary to uveitis may occur by any one or by a combination of several different pathophysiological mechanisms. These include acute angle-closure due to iris bombé caused by posterior synechiae; chronic angle-closure due to peripheral anterior synechiae; and open angle glaucoma due to obstruction and/or inflammation of the trabecular meshwork. Secretory hypotony may mask impairment of outflow, while steroids used to treat the uveitis may further complicate the situation by causing a rise in intraocular pressure. Careful delineation of the pathophysiology involved is the cornerstone of successful management.
葡萄膜炎继发性青光眼可能通过任何一种或几种不同的病理生理机制共同作用而发生。这些机制包括：后粘连导致虹膜膨隆引起的急性闭角；周边前粘连导致的慢性闭角；以及小梁网阻塞和/或炎症引起的开角型青光眼。分泌性低眼压可能掩盖房水流出障碍，而用于治疗葡萄膜炎的类固醇药物可能会导致眼压升高，从而使情况更加复杂。仔细界定所涉及的病理生理学是成功治疗的基石。
