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Type II Toxin-Antitoxin Systems and Persister Cells.

作者信息

Holden David W, Errington Jeff

机构信息

MRC Centre for Molecular Bacteriology and Infection, Imperial College London, London, United Kingdom

Centre for Bacterial Cell Biology, Institute for Cell and Molecular Biosciences, Newcastle University, Newcastle upon Tyne, United Kingdom.

出版信息

mBio. 2018 Sep 25;9(5):e01574-18. doi: 10.1128/mBio.01574-18.

DOI:10.1128/mBio.01574-18
PMID:30254124
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC6156201/
Abstract
摘要

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本文引用的文献

1
Reassessing the Role of Type II Toxin-Antitoxin Systems in Formation of Escherichia coli Type II Persister Cells.重新评估 II 型毒素-抗毒素系统在大肠埃希菌 II 型持留细胞形成中的作用。
mBio. 2018 Jun 12;9(3):e00640-18. doi: 10.1128/mBio.00640-18.
2
Activity of acetyltransferase toxins involved in Salmonella persister formation during macrophage infection.参与沙门氏菌形成持续感染期间巨噬细胞中乙酰转移酶毒素的活性。
Nat Commun. 2018 May 18;9(1):1993. doi: 10.1038/s41467-018-04472-6.
3
(p)ppGpp Controls Bacterial Persistence by Stochastic Induction of Toxin-Antitoxin Activity.(p)ppGpp通过随机诱导毒素-抗毒素活性来控制细菌的持留性。
Cell. 2018 Feb 22;172(5):1135. doi: 10.1016/j.cell.2018.02.023.
4
Prophages and Growth Dynamics Confound Experimental Results with Antibiotic-Tolerant Persister Cells.噬菌体和生长动态使抗生素耐受持久细胞的实验结果复杂化。
mBio. 2017 Dec 12;8(6):e01964-17. doi: 10.1128/mBio.01964-17.
5
A Salmonella Toxin Promotes Persister Formation through Acetylation of tRNA.一种沙门氏菌毒素通过tRNA的乙酰化促进持留菌形成。
Mol Cell. 2016 Jul 7;63(1):86-96. doi: 10.1016/j.molcel.2016.05.002. Epub 2016 Jun 2.
6
Involvement of toxin-antitoxin modules in Burkholderia cenocepacia biofilm persistence.毒素-抗毒素模块在洋葱伯克霍尔德氏菌生物膜持久中的作用。
Pathog Dis. 2014 Aug;71(3):326-35. doi: 10.1111/2049-632X.12177. Epub 2014 Apr 28.
7
Internalization of Salmonella by macrophages induces formation of nonreplicating persisters.巨噬细胞内吞沙门氏菌会诱导非复制持久菌的形成。
Science. 2014 Jan 10;343(6167):204-8. doi: 10.1126/science.1244705.
8
MazF-induced growth inhibition and persister generation in Escherichia coli.MazF 诱导的大肠杆菌生长抑制和持续生存现象的产生。
J Biol Chem. 2014 Feb 14;289(7):4191-205. doi: 10.1074/jbc.M113.510511. Epub 2013 Dec 27.
9
(p)ppGpp controls bacterial persistence by stochastic induction of toxin-antitoxin activity.(p)ppGpp 通过随机诱导毒素-抗毒素活性来控制细菌的持续存在。
Cell. 2013 Aug 29;154(5):1140-1150. doi: 10.1016/j.cell.2013.07.048.
10
Toxin-antitoxin systems are important for niche-specific colonization and stress resistance of uropathogenic Escherichia coli.毒素-抗毒素系统对于尿路致病性大肠杆菌在特定小生境中的定植和应激抵抗非常重要。
PLoS Pathog. 2012;8(10):e1002954. doi: 10.1371/journal.ppat.1002954. Epub 2012 Oct 4.