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骨髓源性巨噬细胞增殖的调控

Regulation of proliferation of bone marrow-derived macrophages.

作者信息

Hume D A, Allan W, Fabrus B, Weidemann M J, Hapel A J, Bartelmez S

出版信息

Lymphokine Res. 1987 Spring;6(2):127-39.

PMID:3035291
Abstract

The rate of mobilisation of macrophages from marrow is an important determinant of the outcome of many pathological lesions. Studies of BMDM have provided insight into the complex feedback regulation that may control macrophage production. In addition BMDM possess several properties in common with transformed cells of other tissue origins and an understanding of their growth regulation may provide an insight into the mechanism of malignant transformation.

摘要

骨髓中巨噬细胞的动员速率是许多病理损伤结果的重要决定因素。对骨髓来源的巨噬细胞（BMDM）的研究为可能控制巨噬细胞产生的复杂反馈调节提供了见解。此外，BMDM具有一些与其他组织来源的转化细胞共有的特性，对其生长调节的理解可能有助于深入了解恶性转化的机制。

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1
Regulation of proliferation of bone marrow-derived macrophages.骨髓源性巨噬细胞增殖的调控
Lymphokine Res. 1987 Spring;6(2):127-39.
2
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[In vivo effect of etoposide on the function of bone marrow-derived macrophages].
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Expression of 20-alpha-hydroxysteroid dehydrogenase in mouse macrophages, hemopoietic cells, and cell lines and its induction by colony-stimulating factors.20-α-羟基类固醇脱氢酶在小鼠巨噬细胞、造血细胞及细胞系中的表达及其受集落刺激因子的诱导作用
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Bone marrow adherent cell hemopoietic growth factor production.骨髓贴壁细胞造血生长因子的产生。
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J Immunol. 1988 Jul 1;141(1):139-44.

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The phosphoproteome of toll-like receptor-activated macrophages.Toll 样受体激活的巨噬细胞的磷酸化蛋白质组。
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Prostaglandin E2 mediates growth arrest in NFS-60 cells by down-regulating interleukin-6 receptor expression.前列腺素E2通过下调白细胞介素-6受体表达介导NFS-60细胞的生长停滞。
Biochem J. 2003 Feb 15;370(Pt 1):315-21. doi: 10.1042/BJ20021512.
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Macrophage heterogeneity occurs through a developmental mechanism.巨噬细胞的异质性通过一种发育机制产生。
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