《适度流体剪切应力可通过激活FAK-MEK5-ERK5-cFos-AP1信号通路调节髓核细胞骨架及周围炎症介质》勘误
Corrigendum to "Moderate Fluid Shear Stress Could Regulate the Cytoskeleton of Nucleus Pulposus and Surrounding Inflammatory Mediators by Activating the FAK-MEK5-ERK5-cFos-AP1 Signaling Pathway".
作者信息
Ye Dongping, Liang Weiguo, Dai Libing, Yao Yicun
机构信息
Guangzhou City Red Cross Hospital, The Fourth Affiliated Hospital of Medical College, Jinan University, Guangzhou 510220, China.
出版信息
Dis Markers. 2018 Sep 27;2018:4597569. doi: 10.1155/2018/4597569. eCollection 2018.
Abstract
[This corrects the article DOI: 10.1155/2018/9405738.].
摘要
[本文更正了文章的数字对象标识符：10.1155/2018/9405738。]
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Corrigendum to "Moderate Fluid Shear Stress Could Regulate the Cytoskeleton of Nucleus Pulposus and Surrounding Inflammatory Mediators by Activating the FAK-MEK5-ERK5-cFos-AP1 Signaling Pathway".《适度流体剪切应力可通过激活FAK-MEK5-ERK5-cFos-AP1信号通路调节髓核细胞骨架及周围炎症介质》勘误
Dis Markers. 2018 Sep 27;2018:4597569. doi: 10.1155/2018/4597569. eCollection 2018.
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Moderate Fluid Shear Stress Could Regulate the Cytoskeleton of Nucleus Pulposus and Surrounding Inflammatory Mediators by Activating the FAK-MEK5-ERK5-cFos-AP1 Signaling Pathway.适度的流体切应力可以通过激活 FAK-MEK5-ERK5-cFos-AP1 信号通路来调节核髓核和周围炎症介质的细胞骨架。
Dis Markers. 2018 Jun 12;2018:9405738. doi: 10.1155/2018/9405738. eCollection 2018.
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本文引用的文献
1
Moderate Fluid Shear Stress Could Regulate the Cytoskeleton of Nucleus Pulposus and Surrounding Inflammatory Mediators by Activating the FAK-MEK5-ERK5-cFos-AP1 Signaling Pathway.适度的流体切应力可以通过激活 FAK-MEK5-ERK5-cFos-AP1 信号通路来调节核髓核和周围炎症介质的细胞骨架。
Dis Markers. 2018 Jun 12;2018:9405738. doi: 10.1155/2018/9405738. eCollection 2018.
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