Modeling Microvillus Inclusion Formation In Vitro.
作者信息
Engevik Amy C, Goldenring James R
机构信息
Department of Surgery, Vanderbilt University School of Medicine, Nashville, Tennessee.
Epithelial Biology Center, Vanderbilt University School of Medicine, Nashville, Tennessee.
出版信息
Cell Mol Gastroenterol Hepatol. 2018 Sep 5;6(4):472-473. doi: 10.1016/j.jcmgh.2018.08.002. eCollection 2018.
Abstract
摘要
相似文献
1
Modeling Microvillus Inclusion Formation In Vitro.体外模拟微绒毛包涵体形成
Cell Mol Gastroenterol Hepatol. 2018 Sep 5;6(4):472-473. doi: 10.1016/j.jcmgh.2018.08.002. eCollection 2018.
2
The zebrafish goosepimples/myosin Vb mutant exhibits cellular attributes of human microvillus inclusion disease.斑马鱼鸡皮疙瘩/肌球蛋白Vb突变体表现出人类微绒毛包涵体病的细胞特征。
Mech Dev. 2016 Nov;142:62-74. doi: 10.1016/j.mod.2016.08.001. Epub 2016 Aug 3.
3
Myosin Vb uncoupling from RAB8A and RAB11A elicits microvillus inclusion disease.肌球蛋白Vb与RAB8A和RAB11A解偶联引发微绒毛包涵体病。
J Clin Invest. 2014 Jul;124(7):2947-62. doi: 10.1172/JCI71651. Epub 2014 Jun 2.
4
Microvillus inclusion disease: loss of Myosin vb disrupts intracellular traffic and cell polarity.微绒毛包涵体病：Myosin vb 的缺失破坏细胞内运输和细胞极性。
Traffic. 2014 Jan;15(1):22-42. doi: 10.1111/tra.12131. Epub 2013 Nov 19.
5
An inducible mouse model for microvillus inclusion disease reveals a role for myosin Vb in apical and basolateral trafficking.一种用于微绒毛包涵体病的诱导型小鼠模型揭示了肌球蛋白Vb在顶端和基底外侧运输中的作用。
Proc Natl Acad Sci U S A. 2015 Oct 6;112(40):12408-13. doi: 10.1073/pnas.1516672112. Epub 2015 Sep 21.
6
Microvillus inclusion disease with novel MYO5B pathogenic variants.具有新型MYO5B致病变体的微绒毛包涵体病
Histopathology. 2021 Jul;79(1):119-121. doi: 10.1111/his.14348. Epub 2021 Apr 14.
7
Myosin Vb and Rab11a regulate phosphorylation of ezrin in enterocytes.肌球蛋白Vb和Rab11a调节肠上皮细胞中埃兹蛋白的磷酸化。
J Cell Sci. 2014 Mar 1;127(Pt 5):1007-17. doi: 10.1242/jcs.137273. Epub 2014 Jan 10.
8
Functional characterization of mutations in the myosin Vb gene associated with microvillus inclusion disease.肌球蛋白 Vb 基因突变与微绒毛包涵体病的功能特征。
J Pediatr Gastroenterol Nutr. 2011 Mar;52(3):307-13. doi: 10.1097/MPG.0b013e3181eea177.
9
MYO5B mutations cause microvillus inclusion disease and disrupt epithelial cell polarity.肌球蛋白Ⅴb（MYO5B）突变导致微绒毛包涵体病并破坏上皮细胞极性。
Nat Genet. 2008 Oct;40(10):1163-5. doi: 10.1038/ng.225. Epub 2008 Aug 24.
10
Identification of intestinal ion transport defects in microvillus inclusion disease.微绒毛包涵体病中肠道离子转运缺陷的鉴定。
Am J Physiol Gastrointest Liver Physiol. 2016 Jul 1;311(1):G142-55. doi: 10.1152/ajpgi.00041.2016. Epub 2016 May 26.
本文引用的文献
1
Dynamic Formation of Microvillus Inclusions During Enterocyte Differentiation in -Deficient Intestinal Organoids.肠上皮细胞分化过程中 - 缺陷肠类器官中微绒毛包含物的动态形成。
Cell Mol Gastroenterol Hepatol. 2018 Aug 14;6(4):477-493.e1. doi: 10.1016/j.jcmgh.2018.08.001. eCollection 2018.
2
Disrupted apical exocytosis of cargo vesicles causes enteropathy in FHL5 patients with Munc18-2 mutations.货物囊泡顶端胞吐作用的破坏导致患有Munc18-2突变的FHL5患者出现肠病。
JCI Insight. 2017 Jul 20;2(14). doi: 10.1172/jci.insight.94564.
3
Loss of MYO5B in mice recapitulates Microvillus Inclusion Disease and reveals an apical trafficking pathway distinct to neonatal duodenum.小鼠中MYO5B的缺失重现了微绒毛包涵体病，并揭示了一条与新生儿十二指肠不同的顶端运输途径。
Cell Mol Gastroenterol Hepatol. 2016 Feb 1;2(2):131-157. doi: 10.1016/j.jcmgh.2015.11.009.
4
Myosin Vb uncoupling from RAB8A and RAB11A elicits microvillus inclusion disease.肌球蛋白Vb与RAB8A和RAB11A解偶联引发微绒毛包涵体病。
J Clin Invest. 2014 Jul;124(7):2947-62. doi: 10.1172/JCI71651. Epub 2014 Jun 2.
5
Loss of syntaxin 3 causes variant microvillus inclusion disease.突触结合蛋白 3 缺失导致变异微绒毛包涵体病。
Gastroenterology. 2014 Jul;147(1):65-68.e10. doi: 10.1053/j.gastro.2014.04.002. Epub 2014 Apr 12.
6
Single Lgr5 stem cells build crypt-villus structures in vitro without a mesenchymal niche.单个Lgr5干细胞在体外无需间充质微环境即可构建隐窝-绒毛结构。
Nature. 2009 May 14;459(7244):262-5. doi: 10.1038/nature07935. Epub 2009 Mar 29.
7
MYO5B mutations cause microvillus inclusion disease and disrupt epithelial cell polarity.肌球蛋白Ⅴb（MYO5B）突变导致微绒毛包涵体病并破坏上皮细胞极性。
Nat Genet. 2008 Oct;40(10):1163-5. doi: 10.1038/ng.225. Epub 2008 Aug 24.
8
Microvillous inclusion disease (microvillous atrophy).微绒毛包涵体病（微绒毛萎缩）
Orphanet J Rare Dis. 2006 Jun 26;1:22. doi: 10.1186/1750-1172-1-22.
9
Interaction of Munc-18-2 with syntaxin 3 controls the association of apical SNAREs in epithelial cells.Munc-18-2与 syntaxin 3 的相互作用控制上皮细胞顶端 SNARE 蛋白的结合。
J Cell Sci. 1998 Sep;111 ( Pt 17):2681-8. doi: 10.1242/jcs.111.17.2681.
Zotero 插件