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超高深度扩增子测序表明,1 型 NF1 缺失患者中不存在正常细胞的低级别镶嵌现象。

Ultra-deep amplicon sequencing indicates absence of low-grade mosaicism with normal cells in patients with type-1 NF1 deletions.

机构信息

Institute of Human Genetics, University of Ulm, Albert-Einstein-Allee 11, 89081, Ulm, Germany.

Department of Neurology, University Hospital Hamburg Eppendorf, 20246, Hamburg, Germany.

出版信息

Hum Genet. 2019 Jan;138(1):73-81. doi: 10.1007/s00439-018-1961-5. Epub 2018 Nov 26.

DOI:10.1007/s00439-018-1961-5
PMID:30478644
Abstract

Different types of large NF1 deletion are distinguishable by breakpoint location and potentially also by the frequency of mosaicism with normal cells lacking the deletion. However, low-grade mosaicism with fewer than 10% normal cells has not yet been excluded for all NF1 deletion types since it is impossible to assess by the standard techniques used to identify such deletions, including MLPA and array analysis. Here, we used ultra-deep amplicon sequencing to investigate the presence of normal cells in the blood of 20 patients with type-1 NF1 deletions lacking mosaicism according to MLPA. The ultra-deep sequencing entailed the screening of 96 amplicons for heterozygous SNVs located within the NF1 deletion region. DNA samples from three previously identified patients with type-2 NF1 deletions and low-grade mosaicism with normal cells as determined by FISH or microsatellite marker analysis were used to validate our methodology. In these type-2 NF1 deletion samples, proportions of 5.3%, 6.6% and 15.0% normal cells, respectively, were detected by ultra-deep amplicon sequencing. However, using this highly sensitive method, none of the 20 patients with type-1 NF1 deletions included in our analysis exhibited low-grade mosaicism with normal cells in blood, thereby supporting the view that the vast majority of type-1 deletions are germline deletions.

摘要

不同类型的大型 NF1 缺失可通过断点位置区分,并且潜在地也可通过正常细胞缺乏缺失的嵌合频率来区分。然而,由于无法通过用于鉴定此类缺失的标准技术(包括 MLPA 和阵列分析)进行评估,尚未排除所有 NF1 缺失类型的低级别嵌合体,其中正常细胞少于 10%。在这里,我们使用超深度扩增子测序来研究 20 名根据 MLPA 无嵌合体的 1 型 NF1 缺失患者血液中正常细胞的存在情况。超深度测序需要筛选位于 NF1 缺失区域内的杂合 SNV 的 96 个扩增子。使用来自三个先前鉴定的 2 型 NF1 缺失患者的 DNA 样本,这些患者通过 FISH 或微卫星标记分析确定为低级别嵌合体,正常细胞,用于验证我们的方法。在这些 2 型 NF1 缺失样本中,通过超深度扩增子测序分别检测到 5.3%、6.6%和 15.0%的正常细胞。然而,使用这种高度敏感的方法,我们分析的 20 名 1 型 NF1 缺失患者中没有一人在血液中表现出低级别嵌合体的正常细胞,从而支持绝大多数 1 型缺失是种系缺失的观点。

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