Ifi204/p204, a new piece in the sepsis puzzle.
作者信息
Subramanian Gayatri, Chakravarti Ritu, Chattopadhyay Saurabh
机构信息
Department of Medical Microbiology and Immunology, University of Toledo College of Medicine and Life Sciences, Toledo, OH, USA.
Department of Physiology and Pharmacology, University of Toledo College of Medicine and Life Sciences, Toledo, OH, USA.
出版信息
Ann Transl Med. 2018 Nov;6(Suppl 1):S12. doi: 10.21037/atm.2018.09.22.
Abstract
摘要
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本文引用的文献
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p204 Is Required for Canonical Lipopolysaccharide-induced TLR4 Signaling in Mice.p204 在小鼠中对脂多糖诱导的 TLR4 信号传导是必需的。
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IFI16 and cGAS cooperate in the activation of STING during DNA sensing in human keratinocytes.IFI16 和 cGAS 在人类角质形成细胞的 DNA 感应过程中协同激活 STING。
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EGFR kinase activity is required for TLR4 signaling and the septic shock response.TLR4信号传导和脓毒性休克反应需要EGFR激酶活性。
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Erlotinib protects against LPS-induced endotoxicity because TLR4 needs EGFR to signal.厄洛替尼可预防脂多糖诱导的内毒素血症，因为Toll样受体4（TLR4）需要表皮生长因子受体（EGFR）来传递信号。
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PRRs are watching you: Localization of innate sensing and signaling regulators.模式识别受体在监视着你：天然免疫感应与信号调节因子的定位
Virology. 2015 May;479-480:104-9. doi: 10.1016/j.virol.2015.02.051. Epub 2015 Mar 20.
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cGAS and Ifi204 cooperate to produce type I IFNs in response to Francisella infection.cGAS和Ifi204协同作用，以响应弗朗西斯菌感染产生I型干扰素。
J Immunol. 2015 Apr 1;194(7):3236-45. doi: 10.4049/jimmunol.1402764. Epub 2015 Feb 20.
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Tyrosine phosphorylation in Toll-like receptor signaling.Toll样受体信号通路中的酪氨酸磷酸化
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Inhibition of viral pathogenesis and promotion of the septic shock response to bacterial infection by IRF-3 are regulated by the acetylation and phosphorylation of its coactivators.IRF-3 的共激活因子的乙酰化和磷酸化调节了其对病毒发病机制的抑制作用和对细菌感染性休克反应的促进作用。
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