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更正：结节性硬化复合物2缺失使肿瘤对奈非那韦-硼替佐米治疗敏感，从而加剧内质网应激诱导的细胞死亡。

Correction: Loss of tuberous sclerosis complex 2 sensitizes tumors to nelfinavir-bortezomib therapy to intensify endoplasmic reticulum stress-induced cell death.

作者信息

Johnson Charlotte E, Dunlop Elaine A, Seifan Sara, McCann Henry D, Hay Trevor, Parfitt Geraint J, Jones Ashley T, Giles Peter J, Shen Ming H, Sampson Julian R, Errington Rachel J, Davies D Mark, Tee Andrew R

机构信息

Division of Cancer and Genetics, Cardiff University, Heath Park, Cardiff, CF14 4XN, UK.

European Cancer Stem Cell Research Institute, Cardiff University, Hadyn Ellis Building, Maindy Road, Cardiff, CF24 4HQ, UK.

出版信息

Oncogene. 2019 Apr;38(16):3102. doi: 10.1038/s41388-018-0598-0.

DOI:10.1038/s41388-018-0598-0
PMID:30622341
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC8075855/
Abstract

This article was originally published under standard licence, but has now been made available under a CC BY 4.0 license. The PDF and HTML versions of the paper have been modified accordingly.

摘要

本文最初以标准许可发布，但现已根据知识共享署名4.0许可提供。论文的PDF和HTML版本已相应修改。

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Correction: Loss of tuberous sclerosis complex 2 sensitizes tumors to nelfinavir-bortezomib therapy to intensify endoplasmic reticulum stress-induced cell death.更正：结节性硬化复合物2缺失使肿瘤对奈非那韦-硼替佐米治疗敏感，从而加剧内质网应激诱导的细胞死亡。
Oncogene. 2019 Apr;38(16):3102. doi: 10.1038/s41388-018-0598-0.
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