Tokunaga T, Kuboyama M, Kojo N, Matsuo H, Shigemori M, Kuramoto S
Department of Neurosurgery, Fuchu Keizinkai Hospital.
No Shinkei Geka. 1988 Nov;16(12):1389-93.
A 61-year-old normotensive woman was admitted to our hospital with complaints of severe headache and nausea. She has had a limp due to the poliomyelitis in her childhood. Three days before admission, she had a headache without unconsciousness. On admission, she was almost alert. Her vital signs were normal. A mild monoparesis of the atrophic right lower extremity was evident. This impairment was due to her past poliomyelitis. There were no other motor palsy, sensory loss, nuchal rigidity, and papilledema. A plain CT scan showed a crescent high density zone adjacent to a round high density mass in the left convexity. A cerebral angiogram showed a vascular left temporoparietal mass supplied by a middle meningeal artery. A craniotomy was then immediately performed. The well encapsulated tumor was totally resected, and the adjacent subdural hematoma was also evacuated. Histologically, the tumor was transitional meningioma with areas containing thin-walled vessels and focal necrosis. Postoperative course was uneventful. The origin of hemorrhage in this case would seem to be the thin-walled blood vessel with loss of vessel support. The clinical significance and the mechanism of the hemorrhage from the meningioma were discussed.
一名61岁血压正常的女性因严重头痛和恶心入院。她童年时因患小儿麻痹症而跛行。入院前三天,她出现头痛但未昏迷。入院时,她神志基本清醒。生命体征正常。右下肢萎缩,有轻度单瘫,这一损伤是由她过去的小儿麻痹症所致。无其他运动麻痹、感觉丧失、颈项强直及视乳头水肿。普通CT扫描显示左脑凸面有一圆形高密度肿块,其旁有一新月形高密度区。脑血管造影显示左侧颞顶叶有一由脑膜中动脉供血滋养的血管性肿块。随后立即进行了开颅手术。完整包膜的肿瘤被完全切除,相邻的硬膜下血肿也被清除。组织学检查显示肿瘤为过渡型脑膜瘤,部分区域有薄壁血管和局灶性坏死。术后病程平稳。该病例出血的原因似乎是薄壁血管失去血管支持。讨论了脑膜瘤出血的临床意义及机制。