Hackenbroch M H, Fischer V, Matzen K
MMW Munch Med Wochenschr. 1978 Jun 9;120(23):795-8.
The etiological assessment of aseptic femoral head necrosis in adults is facilitated by investigation of the uric acid level and of fat metabolism parameters. From 98 of our own patients it appears that femoral head necrosis after trauma, irradiation therapy and caisson disease and after massive doses of cortisone only exceptionally shows pathological serum levels. Femoral head necrosis with manifest metabolic diseases shows 53.3% hyperuricemias and hyperlipemias or dyslipidemias. Of femoral head necroses without concomitant diseases, prior physical effects and administration of cortisone, 91% had hyperuricemia and 65% hyperlipemia or dyslipidemia.
通过对尿酸水平和脂肪代谢参数的检测，有助于对成人无菌性股骨头坏死进行病因评估。从我们自己的98例患者来看，创伤、放射治疗、沉箱病以及大剂量使用可的松后发生的股骨头坏死，仅在极少数情况下会出现血清水平异常。伴有明显代谢疾病的股骨头坏死患者中，53.3%存在高尿酸血症和高脂血症或血脂异常。在无伴随疾病、既往无身体影响且未使用可的松的股骨头坏死患者中，91%有高尿酸血症，65%有高脂血症或血脂异常。
